Disruption of cortical actin in skeletal muscle demonstrates an essential role of the cytoskeleton in glucose transporter 4 translocation in insulin-sensitive tissues.
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Signaling mechanisms of glucose-induced F-actin remodeling in pancreatic islet β cellsAn actin filament population defined by the tropomyosin Tpm3.1 regulates glucose uptakeRac1--a novel regulator of contraction-stimulated glucose uptake in skeletal muscleStretch-stimulated glucose transport in skeletal muscle is regulated by Rac1Rac1 is a novel regulator of contraction-stimulated glucose uptake in skeletal muscleRac1 signaling is required for insulin-stimulated glucose uptake and is dysregulated in insulin-resistant murine and human skeletal muscleSignaling of the p21-activated kinase (PAK1) coordinates insulin-stimulated actin remodeling and glucose uptake in skeletal muscle cells.Role of insulin-dependent cortical fodrin/spectrin remodeling in glucose transporter 4 translocation in rat adipocytesMyo1c binding to submembrane actin mediates insulin-induced tethering of GLUT4 vesiclesArp2/3- and cofilin-coordinated actin dynamics is required for insulin-mediated GLUT4 translocation to the surface of muscle cellsCortactin, an actin binding protein, regulates GLUT4 translocation via actin filament remodelingTropomodulin3 is a novel Akt2 effector regulating insulin-stimulated GLUT4 exocytosis through cortical actin remodeling.Focal Adhesion Kinase contributes to insulin-induced actin reorganization into a mesh harboring Glucose transporter-4 in insulin resistant skeletal muscle cellsChromium enhances insulin responsiveness via AMPK.PKC-induced intracellular trafficking of Ca(V)2 precedes its rapid recruitment to the plasma membrane.Exocytosis mechanisms underlying insulin release and glucose uptake: conserved roles for Munc18c and syntaxin 4Crucial role of the small GTPase Rac1 in insulin-stimulated translocation of glucose transporter 4 to the mouse skeletal muscle sarcolemma.Myo1c regulates glucose uptake in mouse skeletal muscle.Leukemia inhibitory factor regulates trafficking of T-type Ca2+ channels.Subcellular trafficking of the substrate transporters GLUT4 and CD36 in cardiomyocytesLipid-overloaded enlarged adipocytes provoke insulin resistance independent of inflammation.Fat-induced membrane cholesterol accrual provokes cortical filamentous actin destabilisation and glucose transport dysfunction in skeletal muscleRegulation of glucose transport by ROCK1 differs from that of ROCK2 and is controlled by actin polymerizationAMPK enhances insulin-stimulated GLUT4 regulation via lowering membrane cholesterol.Turning signals on and off: GLUT4 traffic in the insulin-signaling highway.Insulin signaling and the regulation of glucose transport.Antidiabetogenic effects of chromium mitigate hyperinsulinemia-induced cellular insulin resistance via correction of plasma membrane cholesterol imbalance."Actin"g on GLUT4: membrane & cytoskeletal components of insulin action.Phosphatidylinositol 4,5-bisphosphate reverses endothelin-1-induced insulin resistance via an actin-dependent mechanismEndothelin-1 impairs glucose transporter trafficking via a membrane-based mechanismLoss of cortical actin filaments in insulin-resistant skeletal muscle cells impairs GLUT4 vesicle trafficking and glucose transport.Putting Rac1 on the path to glucose uptake.Mechanisms for increased myocardial fatty acid utilization following short-term high-fat feeding.Measuring GLUT4 translocation in mature muscle fibers.p21-Activated protein kinases and their emerging roles in glucose homeostasis.Rho GTPases in insulin-stimulated glucose uptake.Molecular mechanisms for the regulation of insulin-stimulated glucose uptake by small guanosine triphosphatases in skeletal muscle and adipocytes.Lysophosphatidic acid-mediated augmentation of cardiomyocyte lipoprotein lipase involves actin cytoskeleton reorganization.Glucose transporters: cellular links to hyperglycemia in insulin resistance and diabetes.Exercise training prevents skeletal muscle plasma membrane cholesterol accumulation, cortical actin filament loss, and insulin resistance in C57BL/6J mice fed a western-style high-fat diet.
P2860
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P2860
Disruption of cortical actin in skeletal muscle demonstrates an essential role of the cytoskeleton in glucose transporter 4 translocation in insulin-sensitive tissues.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@ast
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@en
type
label
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@ast
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@en
prefLabel
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@ast
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@en
P2093
P2860
P356
P1476
Disruption of cortical actin i ...... in insulin-sensitive tissues.
@en
P2093
Andrew B Strawbridge
Eric D Hawkins
Jeffrey S Elmendorf
Joseph T Brozinick
P2860
P304
40699-40706
P356
10.1074/JBC.M402697200
P407
P577
2004-07-06T00:00:00Z