Tumour necrosis factor and PI3-kinase control oestrogen receptor alpha protein level and its transrepression function
about
Extreme growth factor signalling can promote oestrogen receptor-alpha loss: therapeutic implications in breast cancerEffect of macrophages on breast cancer cell proliferation, and on expression of hormone receptors, uPAR and HER-2Inositol polyphosphate 4-phosphatase II regulates PI3K/Akt signaling and is lost in human basal-like breast cancersInterplay between estrogen receptor and AKT in estradiol-induced alternative splicingAPOBEC3B-Mediated Cytidine Deamination Is Required for Estrogen Receptor Action in Breast Cancer.Expression of a phosphorylated p130(Cas) substrate domain attenuates the phosphatidylinositol 3-kinase/Akt survival pathway in tamoxifen resistant breast cancer cells.An Approach to Breast Cancer Immunotherapy: The Apoptotic Activity of Recombinant Anti-Interleukin-6 Monoclonal Antibodies in Intact Tumour Microenvironment of Breast Carcinoma.ERK1/2 inhibition increases antiestrogen treatment efficacy by interfering with hypoxia-induced downregulation of ERalpha: a combination therapy potentially targeting hypoxic and dormant tumor cells.Oncostatin-M promotes phenotypic changes associated with mesenchymal and stem cell-like differentiation in breast cancer.
P2860
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P2860
Tumour necrosis factor and PI3-kinase control oestrogen receptor alpha protein level and its transrepression function
description
2004 nî lūn-bûn
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2004年の論文
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2004年論文
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2004年論文
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name
Tumour necrosis factor and PI3 ...... d its transrepression function
@ast
Tumour necrosis factor and PI3 ...... d its transrepression function
@en
type
label
Tumour necrosis factor and PI3 ...... d its transrepression function
@ast
Tumour necrosis factor and PI3 ...... d its transrepression function
@en
prefLabel
Tumour necrosis factor and PI3 ...... d its transrepression function
@ast
Tumour necrosis factor and PI3 ...... d its transrepression function
@en
P2093
P2860
P356
P1476
Tumour necrosis factor and PI3 ...... d its transrepression function
@en
P2093
M S Marshall
P Bhat-Nakshatri
R A Campbell
T R Newton
P2860
P2888
P304
P356
10.1038/SJ.BJC.6601541
P407
P577
2004-02-01T00:00:00Z