Heterogeneous upregulation of apamin-sensitive potassium currents in failing human ventricles - Wikidata - wikimedia - TriplyDB

Heterogeneous upregulation of apamin-sensitive potassium currents in failing human ventricles

Heterogeneous upregulation of apamin-sensitive potassium currents in failing human ventricles

http://www.wikidata.org/entity/Q36701719

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Ion Channels in the Heart Small-conductance Ca2+ -activated K+ channels and cardiac arrhythmias Amiodarone inhibits apamin-sensitive potassium currents SK channels and ventricular arrhythmias in heart failure Functional interaction with filamin A and intracellular Ca2+ enhance the surface membrane expression of a small-conductance Ca2+-activated K+ (SK2) channel Arrhythmogenic calmodulin mutations impede activation of small-conductance calcium-activated potassium current KCNN2 polymorphisms and cardiac tachyarrhythmias.Apamin does not inhibit human cardiac Na+ current, L-type Ca2+ current or other major K+ currents Comparative normal/failing rat myocardium cell membrane chromatographic analysis system for screening specific components that counteract doxorubicin-induced heart failure from Acontium carmichaeli.Calcium-activated potassium current modulates ventricular repolarization in chronic heart failure.Apamin-sensitive calcium-activated potassium currents in rabbit ventricles with chronic myocardial infarction.Targeting the Small- and Intermediate-Conductance Ca-Activated Potassium Channels: The Drug-Binding Pocket at the Channel/Calmodulin Interface Small conductance calcium-activated potassium current is important in transmural repolarization of failing human ventricles.Patients with Dilated Cardiomyopathy and Sustained Monomorphic Ventricular Tachycardia Show Up-Regulation of KCNN3 and KCNJ2 Genes and CACNG8-Linked Left Ventricular Dysfunction Small-Conductance Calcium-Activated Potassium Current in Normal Rabbit Cardiac Purkinje Cells.Proarrhythmic effect of blocking the small conductance calcium activated potassium channel in isolated canine left atrium.Apamin-sensitive potassium current modulates action potential duration restitution and arrhythmogenesis of failing rabbit ventricles Revisiting K(+) channel-dependent electrical remodeling in the border zone.Critical roles of a small conductance Ca²⁺-activated K⁺ channel (SK3) in the repolarization process of atrial myocytes.Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.TRPM4 in cardiac electrical activity.Modulation of Cardiac Potassium Current by Neural Tone and Ischemia.SK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR.Small-conductance calcium-activated potassium (SK) channels contribute to action potential repolarization in human atria.Rat Models of Ventricular Fibrillation Following Acute Myocardial Infarction.Apamin induces early afterdepolarizations and torsades de pointes ventricular arrhythmia from failing rabbit ventricles exhibiting secondary rises in intracellular calcium Potassium channels in the heart: structure, function and regulation.Structural insights into the potency of SK channel positive modulators.A pharmacologic activator of endothelial KCa channels increases systemic conductance and reduces arterial pressure in an anesthetized pig model.Dynamical effects of calcium-sensitive potassium currents on voltage and calcium alternans.Role of Apamin-Sensitive Calcium-Activated Small-Conductance Potassium Currents on the Mechanisms of Ventricular Fibrillation in Pacing-Induced Failing Rabbit Hearts.Role of apamin sensitive small conductance calcium-activated potassium currents in long term cardiac memory in rabbits.The small neurotoxin apamin blocks not only small conductance Ca2+ activated K+ channels (SK type) but also the voltage dependent Kv1.3 channel.Small-conductance Ca2+-activated K+ channels: insights into their roles in cardiovascular disease.Small-conductance Ca2+-activated K+ current is upregulated via the phosphorylation of CaMKII in cardiac hypertrophy from spontaneously hypertensive rats.Ca2+/Calmodulin-Dependent Protein Kinase II (CaMKII) Increases Small-Conductance Ca2+-Activated K+ Current in Patients with Chronic Atrial Fibrillation.

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Heterogeneous upregulation of apamin-sensitive potassium currents in failing human ventricles

http://www.wikidata.org/entity/Q36701719

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年论文

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Heterogeneous upregulation of ...... ts in failing human ventricles

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Heterogeneous upregulation of ...... ts in failing human ventricles

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JAHA.112.004713

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Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease

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Heterogeneous upregulation of ...... ts in failing human ventricles

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Bich-Lien Nguyen

http://www.w3.org/2001/XMLSchema#string

Isik Turker

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John C Lopshire

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Michael Rubart

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Peng-Sheng Chen

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Po-Cheng Chang

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Saqib Masroor

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Tomohiko Ai

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Wen Tao

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Zhenhui Chen

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P2860

Molecular coupling of a Ca2+-activated K+ channel to L-type Ca2+ channels via alpha-actinin2

Small-conductance, calcium-activated potassium channels from mammalian brain

Epidemiology and risk profile of heart failure

Ablation of a Ca2+-activated K+ channel (SK2 channel) results in action potential prolongation in atrial myocytes and atrial fibrillation

Quantitative expression analysis of the small conductance calcium-activated potassium channels, SK1, SK2 and SK3, in human brain

Small-conductance Ca2+-activated K+ channels: form and function

Differential expression of small-conductance Ca2+-activated K+ channels SK1, SK2, and SK3 in mouse atrial and ventricular myocytes

Loss of T-tubules and other changes to surface topography in ventricular myocytes from failing human and rat heart.

Transmural dispersion of repolarization in failing and nonfailing human ventricle

Electrical remodeling in the failing heart.

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