Excitatory transmission onto AgRP neurons is regulated by cJun NH2-terminal kinase 3 in response to metabolic stress.
about
JNK at the crossroad of obesity, insulin resistance, and cell stress response.Pharmacological Inhibition of c-Jun N-terminal Kinase Reduces Food Intake and Sensitizes Leptin's Anorectic Signaling Actions.Caloric restriction stabilizes body weight and accelerates behavioral recovery in aged rats after focal ischemia.p53 in AgRP neurons is required for protection against diet-induced obesity via JNK1
P2860
Excitatory transmission onto AgRP neurons is regulated by cJun NH2-terminal kinase 3 in response to metabolic stress.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Excitatory transmission onto A ...... response to metabolic stress.
@ast
Excitatory transmission onto A ...... response to metabolic stress.
@en
type
label
Excitatory transmission onto A ...... response to metabolic stress.
@ast
Excitatory transmission onto A ...... response to metabolic stress.
@en
prefLabel
Excitatory transmission onto A ...... response to metabolic stress.
@ast
Excitatory transmission onto A ...... response to metabolic stress.
@en
P2093
P2860
P50
P356
P1433
P1476
Excitatory transmission onto A ...... response to metabolic stress.
@en
P2093
Bradford B Lowell
Caroline Morel
Dae Young Jung
Jason K Kim
Joseph C Madara
Julie Cavanagh-Kyros
Kathryn Chase
Neil Aronin
Norman J Kennedy
Roger J Davis
P2860
P304
P356
10.7554/ELIFE.10031
P407
P577
2016-02-24T00:00:00Z