Model systems for the study of heart development and disease. Cardiac neural crest and conotruncal malformations
about
GATA6 mutations cause human cardiac outflow tract defects by disrupting semaphorin-plexin signalingCoordinating tissue interactions: Notch signaling in cardiac development and diseaseProgramming and reprogramming a human heart cellCapturing structure and function in an embryonic heart with biophotonic tools.D-transposition of the great arteries: the current era of the arterial switch operationDevelopment of the endocardium.Dosage effects of cohesin regulatory factor PDS5 on mammalian development: implications for cohesinopathiesHes1 is expressed in the second heart field and is required for outflow tract developmentTargeted deletion of Hand2 in cardiac neural crest-derived cells influences cardiac gene expression and outflow tract developmentPrdm1 functions in the mesoderm of the second heart field, where it interacts genetically with Tbx1, during outflow tract morphogenesis in the mouse embryoBrn3a regulates the transition from neurogenesis to terminal differentiation and represses non-neural gene expression in the trigeminal ganglionHes1 expression is reduced in Tbx1 null cells and is required for the development of structures affected in 22q11 deletion syndromeA Tbx1-Six1/Eya1-Fgf8 genetic pathway controls mammalian cardiovascular and craniofacial morphogenesisTbx3 is required for outflow tract developmentRequirement for integrin-linked kinase in neural crest migration and differentiation and outflow tract morphogenesisBMP4 is required in the anterior heart field and its derivatives for endocardial cushion remodeling, outflow tract septation, and semilunar valve developmentSkeletogenic fate of zebrafish cranial and trunk neural crest.Outflow tract cushions perform a critical valve-like function in the early embryonic heart requiring BMPRIA-mediated signaling in cardiac neural crestMacondo crude oil from the Deepwater Horizon oil spill disrupts specific developmental processes during zebrafish embryogenesis.Neural crest invasion is a spatially-ordered progression into the head with higher cell proliferation at the migratory front as revealed by the photoactivatable protein, KikGREmbryonic aortic arch hemodynamics are a functional biomarker for ethanol-induced congenital heart defects [Invited]Conotruncal heart defects and common variants in maternal and fetal genes in folate, homocysteine, and transsulfuration pathways.Trigenic neural crest-restricted Smad7 over-expression results in congenital craniofacial and cardiovascular defects.Dicer activity in neural crest cells is essential for craniofacial organogenesis and pharyngeal arch artery morphogenesis.Cranial neural crest migration: new rules for an old road.Novel migrating mouse neural crest cell assay system utilizing P0-Cre/EGFP fluorescent time-lapse imaging.A retinoic acid responsive Hoxa3 transgene expressed in embryonic pharyngeal endoderm, cardiac neural crest and a subdomain of the second heart field.The chromatin remodeling protein CHD7, mutated in CHARGE syndrome, is necessary for proper craniofacial and tracheal developmentVariants of folate metabolism genes and the risk of conotruncal cardiac defects.Jun is required in Isl1-expressing progenitor cells for cardiovascular developmentTwist1 controls a cell-specification switch governing cell fate decisions within the cardiac neural crestSmooth muscle cells from the anastomosed artery are the major precursors for neointima formation in both artery and vein graftsCardiac neural crest is dispensable for outflow tract septation in XenopusPax3 is essential for normal cardiac neural crest morphogenesis but is not required during migration nor outflow tract septation.Evaluating the role of connexin43 in congenital heart disease: Screening for mutations in patients with outflow tract anomalies and the analysis of knock-in mouse modelsMutations of TFAP2B in congenital heart disease patients in Mysore, South India.Notch pathway regulation of neural crest cell development in vivo.Heparan Sulfate Biosynthesis Enzyme, Ext1, Contributes to Outflow Tract Development of Mouse Heart via Modulation of FGF Signaling.The secondary heart field is a new site of calcineurin/Nfatc1 signaling for semilunar valve development.Bioengineering heart muscle: a paradigm for regenerative medicine
P2860
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P2860
Model systems for the study of heart development and disease. Cardiac neural crest and conotruncal malformations
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Model systems for the study of ...... and conotruncal malformations
@ast
Model systems for the study of ...... and conotruncal malformations
@en
type
label
Model systems for the study of ...... and conotruncal malformations
@ast
Model systems for the study of ...... and conotruncal malformations
@en
prefLabel
Model systems for the study of ...... and conotruncal malformations
@ast
Model systems for the study of ...... and conotruncal malformations
@en
P2860
P1476
Model systems for the study of ...... and conotruncal malformations
@en
P2093
Margaret L Kirby
Mary R Hutson
P2860
P304
P356
10.1016/J.SEMCDB.2006.12.004
P577
2006-12-19T00:00:00Z