Ketamine enhances human neural stem cell proliferation and induces neuronal apoptosis via reactive oxygen species-mediated mitochondrial pathway - Wikidata - wikimedia - TriplyDB

Ketamine enhances human neural stem cell proliferation and induces neuronal apoptosis via reactive oxygen species-mediated mitochondrial pathway

Ketamine enhances human neural stem cell proliferation and induces neuronal apoptosis via reactive oxygen species-mediated mitochondrial pathway

http://www.wikidata.org/entity/Q36711438

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Propofol-Induced Neurotoxicity in the Fetal Animal Brain and Developments in Modifying These Effects-An Updated Review of Propofol Fetal Exposure in Laboratory Animal Studies Importance of being Nernst: Synaptic activity and functional relevance in stem cell-derived neurons Modeling anesthetic developmental neurotoxicity using human stem cells Protective Effect of Minocycline Against Ketamine-Induced Injury in Neural Stem Cell: Involvement of PI3K/Akt and Gsk-3 Beta Pathway.Subclinical carbon monoxide limits apoptosis in the developing brain after isoflurane exposure Ketamine exposure in early development impairs specification of the primary germ cell layers.Anaesthetics-induced neurotoxicity in developing brain: an update on preclinical evidence.Emerging model in anesthetic developmental neurotoxicity: human stem cells.Down-regulation of microRNA-21 is involved in the propofol-induced neurotoxicity observed in human stem cell-derived neurons Ketamine causes mitochondrial dysfunction in human induced pluripotent stem cell-derived neurons.Perils of paediatric anaesthesia and novel molecular approaches: An evidence-based review.MicroRNAs: New Players in Anesthetic-Induced Developmental Neurotoxicity Anesthetic Ketamine-Induced DNA Damage in Different Cell Types In Vivo.Carbon monoxide modulates cytochrome oxidase activity and oxidative stress in the developing murine brain during isoflurane exposure.Developmental neurotoxicity screening using human embryonic stem cells.Altered Mitochondrial Dynamics Contributes to Propofol-induced Cell Death in Human Stem Cell-derived Neurons.The role of anesthetic drugs in liver apoptosis.Protective Effect of Antioxidants on Neuronal Dysfunction and Plasticity in Huntington's Disease.Effect of intravenous patient controlled ketamine analgesiaon postoperative pain in opium abusers.DL-2-amino-3-phosphonopropionic acid protects primary neurons from oxygen-glucose deprivation induced injury.Involvement of Cold Inducible RNA-Binding Protein in Severe Hypoxia-Induced Growth Arrest of Neural Stem Cells In Vitro.Stem cells and stem cell-derived tissues and their use in safety assessment.Oxidative Stress and Adult Neurogenesis.Ketamine: A Review of Clinical Pharmacokinetics and Pharmacodynamics in Anesthesia and Pain Therapy.Recent Insights Into Molecular Mechanisms of Propofol-Induced Developmental Neurotoxicity: Implications for the Protective Strategies.Neurogenesis and developmental anesthetic neurotoxicity.Extremely low frequency magnetic fields regulate differentiation of regulatory T cells: Potential role for ROS-mediated inhibition on AKT.Carbon monoxide and anesthesia-induced neurotoxicity.Status of pediatric anesthesiology fellowship research education in the United States: a survey of fellowship program directors.Pink1 attenuates propofol-induced apoptosis and oxidative stress in developing neurons.Biomarkers, Genetics, and Epigenetic Studies to Explore the Neurocognitive Effects of Anesthesia in Children.Effects of Propofol Treatment in Neural Progenitors Derived from Human-Induced Pluripotent Stem Cells.Modulation of Neocortical Development by Early Neuronal Activity: Physiology and Pathophysiology.To Treat or Not to Treat: The Effects of Pain on Experimental Parameters.Insufficient Astrocyte-Derived Brain-Derived Neurotrophic Factor Contributes to Propofol-Induced Neuron Death Through Akt/Glycogen Synthase Kinase 3β/Mitochondrial Fission Pathway.Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos.Carbon monoxide incompletely prevents isoflurane-induced defects in murine neurodevelopment.Ketamine is a neurotoxic agent that could adversely affect the brains of preterm babies.Administration of Ketamine Causes Autophagy and Apoptosis in the Rat Fetal Hippocampus and in PC12 Cells.Ketamine induces apoptosis in lung adenocarcinoma cells by regulating the expression of CD69.

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P2860

Ketamine enhances human neural stem cell proliferation and induces neuronal apoptosis via reactive oxygen species-mediated mitochondrial pathway

http://www.wikidata.org/entity/Q36711438

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Ketamine enhances human neural ...... mediated mitochondrial pathway

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Anesthesia & Analgesia

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Ketamine enhances human neural ...... mediated mitochondrial pathway

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Chika Kikuchi

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Ivan Zaja

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John A Corbett

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Maria Y Muravyeva

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Scott Canfield

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Xiaowen Bai

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Yasheng Yan

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Zeljko J Bosnjak

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P2860

Mechanism of neuroprotective mitochondrial remodeling by PKA/AKAP1

Differentiation of human embryonic stem cells to cardiomyocytes for in vitro and in vivo applications

Ketamine-induced neuronal cell death in the perinatal rhesus monkey

Reduced basal autophagy and impaired mitochondrial dynamics due to loss of Parkinson's disease-associated protein DJ-1

The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis

Calcium, ATP, and ROS: a mitochondrial love-hate triangle

Early exposure to common anesthetic agents causes widespread neurodegeneration in the developing rat brain and persistent learning deficits.

The abolishment of anesthesia-induced cognitive impairment by timely protection of mitochondria in the developing rat brain: the importance of free oxygen radicals and mitochondrial integrity

Potential of ketamine and midazolam, individually or in combination, to induce apoptotic neurodegeneration in the infant mouse brain.

Anesthetics rapidly promote synaptogenesis during a critical period of brain development.

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