BMS-345541 sensitizes MCF-7 breast cancer cells to ionizing radiation by selective inhibition of homologous recombinational repair of DNA double-strand breaks.
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MicroRNA and signal transduction pathways in tumor radiation responseInterplay of Rad51 with NF-κB pathway stimulates expression of HIV-1.Inhibition of mTOR sensitizes breast cancer stem cells to radiation-induced repression of self-renewal through the regulation of MnSOD and Akt.Optimal treatment scheduling of ionizing radiation and sunitinib improves the antitumor activity and allows dose reduction
P2860
BMS-345541 sensitizes MCF-7 breast cancer cells to ionizing radiation by selective inhibition of homologous recombinational repair of DNA double-strand breaks.
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2012 nî lūn-bûn
@nan
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
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name
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@ast
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@en
type
label
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@ast
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@en
prefLabel
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@ast
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@en
P2093
P2860
P356
P1433
P1476
BMS-345541 sensitizes MCF-7 br ...... r of DNA double-strand breaks.
@en
P2093
Daohong Zhou
Jianhui Chang
Junru Wang
Junying Zheng
Lijian Shao
Martin Hauer-Jensen
P2860
P304
P356
10.1667/RR3034.1
P577
2012-12-21T00:00:00Z