Inactivation of the cellular p53 gene is a common feature of Friend virus-induced erythroleukemia: relationship of inactivation to dominant transforming alleles.
about
Fli-1, an Ets-related transcription factor, regulates erythropoietin-induced erythroid proliferation and differentiation: evidence for direct transcriptional repression of the Rb gene during differentiation.Immunity to retroviral infection: the Friend virus modelp45(NFE2) is a negative regulator of erythroid proliferation which contributes to the progression of Friend virus-induced erythroleukemiasAnti-retroviral effects of type I IFN subtypes in vivoWild type p53 gene causes reorganization of cytoskeleton and, therefore, the impaired deformability and difficult migration of murine erythroleukemia cells.Protection against establishment of retroviral persistence by vaccination with a live attenuated virus.Characterization of a live-attenuated retroviral vaccine demonstrates protection via immune mechanisms.Lymphocyte deficiencies increase susceptibility to friend virus-induced erythroleukemia in Fv-2 genetically resistant miceKinetics of the development of protective immunity in mice vaccinated with a live attenuated retrovirus.Role of interleukin-4 (IL-4), IL-12, and gamma interferon in primary and vaccine-primed immune responses to Friend retrovirus infectionThe transforming and suppressor functions of p53 alleles: effects of mutations that disrupt phosphorylation, oligomerization and nuclear translocation.P53 inhibition exacerbates late-stage anthracycline cardiotoxicity.Functional interference between the Spi-1/PU.1 oncoprotein and steroid hormone or vitamin receptors.GATA-1 directly regulates p21 gene expression during erythroid differentiationTemporal effects of gamma interferon deficiency on the course of Friend retrovirus infection in miceFriend Spleen Focus-Forming Virus Activates the Tyrosine Kinase sf-Stk and the Transcription Factor PU.1 to Cause a Multi-Stage Erythroleukemia in Mice.Reprogramming leukemia cells to terminal differentiation and growth arrest by RNA interference of PU.1.Human endogenous retroviruses: nature, occurrence, and clinical implications in human disease.c-myc and bcl-2 modulate p53 function by altering p53 subcellular trafficking during the cell cycle.Suppression of acute anti-friend virus CD8+ T-cell responses by coinfection with lactate dehydrogenase-elevating virusSpi-1/PU.1 transgenic mice develop multistep erythroleukemias.Apoptosis in erythroid progenitors deprived of erythropoietin occurs during the G1 and S phases of the cell cycle without growth arrest or stabilization of wild-type p53.Cell cycle analysis of p53-induced cell death in murine erythroleukemia cells.Growth suppression of Friend virus-transformed erythroleukemia cells by p53 protein is accompanied by hemoglobin production and is sensitive to erythropoietin.Expression of wild-type p53 is not compatible with continued growth of p53-negative tumor cells.Mutant p53 can substitute for human papillomavirus type 16 E6 in immortalization of human keratinocytes but does not have E6-associated trans-activation or transforming activity.Retroviral insertions downstream of the heterogeneous nuclear ribonucleoprotein A1 gene in erythroleukemia cells: evidence that A1 is not essential for cell growth.Mutant p53 tumor suppressor alleles release ras-induced cell cycle growth arrest.Large-scale identification of disease genes involved in acute myeloid leukemiaMulti-stage Friend murine erythroleukemia: molecular insights into oncogenic cooperation.Expression from the murine p53 promoter is mediated by factor binding to a downstream helix-loop-helix recognition motifMutation of the endogenous p53 gene in cells transformed by HPV-16 E7 and EJ c-ras confers a growth advantage involving an autocrine mechanism.Humoral immunity in the Friend retrovirus infection model.Differentiation arrest and stromal cell-independent growth of murine erythroleukemia cells are associated with elevated expression of ets-related genes but not with mutation of p53.Expression of the transcription factor, Spi-1 (PU.1), in differentiating murine erythroleukemia cells Is regulated post-transcriptionally. Evidence for differential stability of transcription factor mRNAs following inducer exposure.Selection for loss of p53 function in T-cell lymphomagenesis is alleviated by Moloney murine leukemia virus infection in myc transgenic miceMoloney murine leukemia virus-induced lymphomas in p53-deficient mice: overlapping pathways in tumor development?Estrogen-induced apoptosis by inhibition of the erythroid transcription factor GATA-1.Genetic determinants of feline leukemia virus-induced lymphoid tumors: patterns of proviral insertion and gene rearrangement.Modulation of immortalizing properties of human papillomavirus type 16 E7 by p53 expression.
P2860
Q24517781-812A2F95-FAA5-40CB-977B-10514113DD9CQ28243606-9C44A5B0-3527-4C01-86C1-6237F6FAC8E8Q28507068-C435A2C4-CE09-468D-8D1C-B47DD2A643ADQ28585391-A173D167-0273-472A-8B62-82F680478E70Q31152518-F5578A71-24CC-446F-8EF1-D63C5EBB9EA0Q33646493-E233163A-5788-4FEA-AE9B-25E944EE62B4Q33783464-4EF0CB1A-07CA-4B7F-B782-A2EC22CC0451Q33816934-80C3D6A2-B6D3-4258-B6AE-09D3913A8997Q33820285-ACACE74E-EEDC-4AEB-98B6-B12E87870D56Q33835324-27692455-8A10-430A-B430-7FB3CA6BA02AQ34045809-EB9B69B4-9E44-46CD-B213-60435029FE8FQ34048273-EEEC25FB-B027-48E6-9387-4ADE20051F91Q34064825-C1D95543-3656-4E2E-87BF-486E16EA3738Q34482319-533FDC51-5877-46E6-9BD7-4ADDD70D47F4Q34969705-B22157A8-007E-4299-A7D2-0CB36EBE7E62Q35260352-E7D88DED-0FCD-483E-990F-7DB118177D45Q35305033-8D8CA12C-D0EA-4B3F-8512-5D6D08CC77EDQ35369410-71DD09C6-CDD3-4A91-A27A-CA71B3CE758CQ35538874-5B91B38F-CA57-481A-8DF2-2E7701DF22B1Q36424006-4925D16D-FE15-44CC-B652-08B5C570D530Q36559604-FA1DA39E-1238-463A-BAEA-9EC73708DCC6Q36655460-7510F306-C87A-4936-9618-6E8D7850057FQ36660017-A0B7587E-1BFB-4047-A1FB-0A83D04BBF15Q36675415-B5BDDFF5-6032-4B9C-93A6-5AE2BB48011FQ36679234-81E32E6E-F0E4-46D2-ADA0-77B9465669DDQ36698822-71B1C241-47B1-4D1B-9C0B-11C32BBBF5D0Q36703019-F4A8DCA4-F392-44E2-9B29-F9849BAA0E93Q36958607-A125F35E-23C1-4F21-8002-30797E07C2F2Q36959992-DE8A0CE9-04B6-469D-AC75-94A35A169BE5Q37317203-9D2D8242-C5A8-4968-B293-D02868D63E06Q37509715-6D1A7E1B-4737-47DB-B5C7-081A73BAE7C3Q37629482-304A4039-51F9-41DA-BCD5-738D5F59CFF8Q37730925-BA5F8A96-9198-42B8-BC3E-FC13141719CEQ38316464-9FC42519-6DF2-4AF3-906C-DEAD251E560AQ38361520-F7E27D41-27CF-4B53-9D03-CFED4907BB81Q39604422-DCADCD95-EAA3-4D2E-B197-DDFA243E74FBQ39873940-8643CF9C-FB8E-4156-A349-9597E19C1FA1Q40018584-11AE6F99-E789-4B1A-AE00-F975D537EE0CQ40043158-6C0322D0-2B80-40E1-B3F0-6C3A34F9A051Q40062749-677DA2A8-7E8D-4D5F-B1CF-49D8DF1DE94F
P2860
Inactivation of the cellular p53 gene is a common feature of Friend virus-induced erythroleukemia: relationship of inactivation to dominant transforming alleles.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Inactivation of the cellular p ...... dominant transforming alleles.
@ast
Inactivation of the cellular p ...... dominant transforming alleles.
@en
type
label
Inactivation of the cellular p ...... dominant transforming alleles.
@ast
Inactivation of the cellular p ...... dominant transforming alleles.
@en
prefLabel
Inactivation of the cellular p ...... dominant transforming alleles.
@ast
Inactivation of the cellular p ...... dominant transforming alleles.
@en
P2093
P2860
P356
P1476
Inactivation of the cellular p ...... dominant transforming alleles.
@en
P2093
Benchimol S
Peacock JW
P2860
P304
P356
10.1128/MCB.10.7.3307
P407
P577
1990-07-01T00:00:00Z