The spinal pathophysiology of spasticity--from a basic science point of view.
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Recovery of neuronal and network excitability after spinal cord injury and implications for spasticityCharacterization of inducible models of Tay-Sachs and related diseaseSpasticity, Motor Recovery, and Neural Plasticity after Stroke.Correlation of Resting Elbow Angle with Spasticity in Chronic Stroke Survivors.New insights into the pathophysiology of post-stroke spasticity.Development of a Haptic Elbow Spasticity Simulator (HESS) for improving accuracy and reliability of clinical assessment of spasticity.Milestones in clinical neurophysiology.Upward synaptic scaling is dependent on neurotransmission rather than spiking.Transcriptional regulation of gene expression clusters in motor neurons following spinal cord injuryProprioceptive neuropathy affects normalization of the H-reflex by exercise after spinal cord injury.Weakened rate-dependent depression of Hoffmann's reflex and increased motoneuron hyperactivity after motor cortical infarction in mice.Modification of spasticity by transcutaneous spinal cord stimulation in individuals with incomplete spinal cord injury.Longitudinal estimation of intramuscular Tibialis Anterior coherence during subacute spinal cord injury: relationship with neurophysiological, functional and clinical outcome measures.Treatment of rat spinal cord injury with the neurotrophic factor albumin-oleic acid: translational application for paralysis, spasticity and pain.The relation between neuromechanical parameters and Ashworth score in stroke patientsClinical understanding of spasticity: implications for practice.Wind-up of stretch reflexes as a measure of spasticity in chronic spinalized rats: The effects of passive exercise and modafinil.Activity-dependent increase in neurotrophic factors is associated with an enhanced modulation of spinal reflexes after spinal cord injury.Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.Down-regulation of KCC2 expression and phosphorylation in motoneurons, and increases the number of in primary afferent projections to motoneurons in mice with post-stroke spasticity.Quantification of the Trömner signs: a sensitive marker for cervical spondylotic myelopathyDendritic spine dysgenesis contributes to hyperreflexia after spinal cord injury.Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptorsKinematic improvement following Botulinum Toxin-A injection in upper-limb spasticity due to strokeIntensity sensitive modulation effect of theta burst form of median nerve stimulation on the monosynaptic spinal reflex.Modulation of inhibitory strength and kinetics facilitates regulation of persistent inward currents and motoneuron excitability following spinal cord injuryIncreased spinal reflex excitability is associated with enhanced central activation during voluntary lengthening contractions in human spinal cord injury.Biceps femoris late latency responses and the "notching sign" in spasticity.Neurophysiological characterization of motor recovery in acute spinal cord injury.Botulinum Neurotoxin A injections influence stretching of the gastrocnemius muscle-tendon unit in an animal model.Selective corticospinal tract injury in the rat induces primary afferent fiber sprouting in the spinal cord and hyperreflexiaOperant conditioning of a spinal reflex can improve locomotion after spinal cord injury in humans.Reciprocal inhibition post-stroke is related to reflex excitability and movement abilityMuscle paresis and passive stiffness: key determinants in limiting function in Hereditary and Sporadic Spastic Paraparesis.The effects of anodal transcranial direct current stimulation and patterned electrical stimulation on spinal inhibitory interneurons and motor function in patients with spinal cord injury.Elbow spasticity during passive stretch-reflex: clinical evaluation using a wearable sensor system.Accuracy and reliability of haptic spasticity assessment using HESS (Haptic Elbow Spasticity Simulator).Haptic recreation of elbow spasticity.Motor cortex bilateral motor representation depends on subcortical and interhemispheric interactions.Muscle weakness and lack of reflex gain adaptation predominate during post-stroke posture control of the wrist.
P2860
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P2860
The spinal pathophysiology of spasticity--from a basic science point of view.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
The spinal pathophysiology of spasticity--from a basic science point of view.
@ast
The spinal pathophysiology of spasticity--from a basic science point of view.
@en
type
label
The spinal pathophysiology of spasticity--from a basic science point of view.
@ast
The spinal pathophysiology of spasticity--from a basic science point of view.
@en
prefLabel
The spinal pathophysiology of spasticity--from a basic science point of view.
@ast
The spinal pathophysiology of spasticity--from a basic science point of view.
@en
P2093
P1433
P1476
The spinal pathophysiology of spasticity--from a basic science point of view.
@en
P2093
P304
P356
10.1111/J.1748-1716.2006.01652.X
P577
2007-02-01T00:00:00Z