Role of alpha-fetoprotein regulatory elements in transcriptional activation in transient heterokaryons
about
Reprogramming towards pluripotency requires AID-dependent DNA demethylationNuclear reprogramming in heterokaryons is rapid, extensive, and bidirectionalActive tissue-specific DNA demethylation conferred by somatic cell nuclei in stable heterokaryonsFoxA proteins regulate H19 endoderm enhancer E1 and exhibit developmental changes in enhancer binding in vivoMouse alpha-fetoprotein gene 5' regulatory elements are required for postnatal regulation by raf and RifMultiple features contribute to efficient constitutive splicing of an unusually large exonNuclear reprogramming to a pluripotent state by three approaches.An RNA polymerase pause site is associated with the immunoglobulin mus poly(A) siteTransdifferentiation and nuclear reprogramming in hematopoietic development and neoplasia.Multiple features contribute to the use of the immunoglobulin M secretion-specific poly(A) signal but are not required for developmental regulation.Tg(Afp-GFP) expression marks primitive and definitive endoderm lineages during mouse development.Recessive mutations in a common pathway block thymocyte apoptosis induced by multiple signals.Cell fusion and plasticity.Regulated immunoglobulin (Ig) RNA processing does not require specific cis-acting sequences: non-Ig RNA can be alternatively processed in B cells and plasma cells.Transactivation of pancreas-specific gene sequences in somatic cell hybridsPosition-dependent activity of alpha -fetoprotein enhancer element III in the adult liver is due to negative regulation.PDGF-A promoter and enhancer elements provide efficient and selective antineoplastic gene therapy in multiple cancer typesPericentral activity of alpha-fetoprotein enhancer 3 and glutamine synthetase upstream enhancer in the adult liver are regulated by β-catenin in mice.Afp::mCherry, a red fluorescent transgenic reporter of the mouse visceral endoderm.Individual mouse alpha-fetoprotein enhancer elements exhibit different patterns of tissue-specific and hepatic position-dependent activitiesEndogenous and transfected mouse alpha-fetoprotein genes in undifferentiated F9 cells are activated in transient heterokaryons.Heterokaryons of cardiac myocytes and fibroblasts reveal the lack of dominance of the cardiac muscle phenotype.The mouse alpha-fetoprotein promoter is repressed in HepG2 hepatoma cells by hepatocyte nuclear factor-3 (FOXA).The "occlusis" model of cell fate restriction.
P2860
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P2860
Role of alpha-fetoprotein regulatory elements in transcriptional activation in transient heterokaryons
description
1990 nî lūn-bûn
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1990年の論文
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1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@ast
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@en
type
label
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@ast
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@en
prefLabel
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@ast
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@en
P2860
P356
P1476
Role of alpha-fetoprotein regu ...... ion in transient heterokaryons
@en
P2093
S M Tilghman
P2860
P304
P356
10.1128/MCB.10.10.5047
P407
P577
1990-10-01T00:00:00Z