Evidence for insulin suppression of baseline luteinizing hormone in women with polycystic ovarian syndrome and normal women. - Wikidata - wikimedia - TriplyDB

Evidence for insulin suppression of baseline luteinizing hormone in women with polycystic ovarian syndrome and normal women.

Evidence for insulin suppression of baseline luteinizing hormone in women with polycystic ovarian syndrome and normal women.

http://www.wikidata.org/entity/Q36732717

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Rescue of obesity-induced infertility in female mice due to a pituitary-specific knockout of the insulin receptor.Insulin resistance and the polycystic ovary syndrome revisited: an update on mechanisms and implications.Lack of association of INS VNTR polymorphism with polycystic ovary syndrome: a meta-analysis Insulin: its role in the central control of reproduction The regulation of reproductive neuroendocrine function by insulin and insulin-like growth factor-1 (IGF-1).Evidence that obesity and androgens have independent and opposing effects on gonadotropin production from puberty to maturity.The circadian variation in Anti-Müllerian hormone in patients with polycystic ovary syndrome differs significantly from normally ovulating women.GnRH Regulates Gonadotropin Gene Expression Through NADPH/Dual Oxidase-Derived Reactive Oxygen Species.Reproductive tissues maintain insulin sensitivity in diet-induced obesity.Trend of Cardio-Metabolic Risk Factors in Polycystic Ovary Syndrome: A Population-Based Prospective Cohort Study.Metabolic and hormonal changes induced by pioglitazone in polycystic ovary syndrome: a randomized, placebo-controlled clinical trial.Blunted sleep-related luteinizing hormone rise in healthy premenarcheal pubertal girls with elevated body mass index.Insulin augments gonadotropin-releasing hormone induction of translation in LbetaT2 cells.The Pathogenesis of Polycystic Ovary Syndrome (PCOS): The Hypothesis of PCOS as Functional Ovarian Hyperandrogenism Revisited.Acute recapitulation of the hyperinsulinemia and hyperlipidemia characteristic of metabolic syndrome suppresses gonadotropins.Reactive Oxygen Species Link Gonadotropin-Releasing Hormone Receptor Signaling Cascades in the Gonadotrope.Induction of Stress Signaling In Vitro and Suppression of Gonadotropin Secretion by Free Fatty Acids in Female Mouse Gonadotropes.Prevalence and impact of hyperandrogenemia in 1,218 women with polycystic ovary syndrome.Symptom patterns and phenotypic subgrouping of women with polycystic ovary syndrome: association between endocrine characteristics and metabolic aberrations

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P2860

Evidence for insulin suppression of baseline luteinizing hormone in women with polycystic ovarian syndrome and normal women.

http://www.wikidata.org/entity/Q36732717

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Evidence for insulin suppressi ...... ian syndrome and normal women.

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Evidence for insulin suppressi ...... ian syndrome and normal women.

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Evidence for insulin suppressi ...... ian syndrome and normal women.

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Evidence for insulin suppressi ...... ian syndrome and normal women.

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Evidence for insulin suppressi ...... ian syndrome and normal women.

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Evidence for insulin suppressi ...... ian syndrome and normal women.

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The Journal of Clinical Endocrinology and Metabolism

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Evidence for insulin suppressi ...... ian syndrome and normal women.

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P2093

Ketan Patel

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Mark A Lawson

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Pamela J Malcolm

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R Jeffrey Chang

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Shelly Sun

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Sonia Jain

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P2860

Polycystic ovarian syndrome: evidence that flutamide restores sensitivity of the gonadotropin-releasing hormone pulse generator to inhibition by estradiol and progesterone.

The origins and sequelae of abnormal neuroendocrine function in polycystic ovary syndrome.

Lack of effect of short-term diazoxide administration on luteinizing hormone secretion in women with polycystic ovary syndrome.

Polycystic ovary syndrome: evidence for reduced sensitivity of the gonadotropin-releasing hormone pulse generator to inhibition by estradiol and progesterone.

Revised 2003 consensus on diagnostic criteria and long-term health risks related to polycystic ovary syndrome (PCOS).

Metformin therapy in polycystic ovary syndrome reduces hyperinsulinemia, insulin resistance, hyperandrogenemia, and systolic blood pressure, while facilitating normal menses and pregnancy.

Insulin administration alters gonadal steroid metabolism independent of changes in gonadotropin secretion in insulin-resistant women with the polycystic ovary syndrome.

Characterization of the inappropriate gonadotropin secretion in polycystic ovary syndrome.

Energy balance and reproduction.

Insulin augments GnRH-stimulated LHbeta gene expression by Egr-1

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2089-2096

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10.1210/JC.2007-2656

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2008-03-11T00:00:00Z

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5516897

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18334581

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2435648

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