Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection. - Wikidata - wikimedia - TriplyDB

Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection.

Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection.

http://www.wikidata.org/entity/Q36737913

about

Human bocavirus: Current knowledge and future challenges Treating the host response to emerging virus diseases: lessons learned from sepsis, pneumonia, influenza and Ebola.Highly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.Inflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections.FXYD5 Is an Essential Mediator of the Inflammatory Response during Lung Injury.Host Response Comparison of H1N1- and H5N1-Infected Mice Identifies Two Potential Death Mechanisms.Involvement of Cytokines in the Pathogenesis of Salt and Water Imbalance in Congestive Heart Failure.Human CD8(+) T Cells Damage Non-infected Epithelial Cells During Influenza Virus Infection In Vitro.Influenza leaves a TRAIL to pulmonary edema.Lung remodeling associated with recovery from acute lung injury.Pathogenesis of severe pneumonia: advances and knowledge gaps.The Impact of the Interferon/TNF-Related Apoptosis-Inducing Ligand Signaling Axis on Disease Progression in Respiratory Viral Infection and Beyond.Influenza virus infection alters ion channel function of Airway and alveolar cells: Mechanisms and physiological sequelae.The Processes and Mechanisms of Cardiac and Pulmonary Fibrosis.Immune responses in influenza A virus and human coronavirus infections: an ongoing battle between the virus and host.Cytokine-Ion Channel Interactions in Pulmonary Inflammation.Depletion of circulating monocytes suppresses IL-17 and HMGB1 expression in mice with LPS-induced acute lung injury.Lymphatic Vessels: The Next Frontier in Lung Transplant.Developing TRAIL/TRAIL death receptor-based cancer therapies.Extracellular Vesicle: An Emerging Mediator of Intercellular Crosstalk in Lung Inflammation and Injury.Divergent Roles for TRAIL in Lung Diseases The Antiviral Effects of Na,K-ATPase Inhibition: A Minireview TRAIL (CD253) Sensitizes Human Airway Epithelial Cells to Toxin-Induced Cell Death Inflammation and Pneumonia: Why Are Some More Susceptible than Others?

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P2860

Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection.

http://www.wikidata.org/entity/Q36737913

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Macrophage-epithelial paracrin ...... ce during influenza infection.

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Macrophage-epithelial paracrin ...... ce during influenza infection.

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Macrophage-epithelial paracrin ...... ce during influenza infection.

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Macrophage-epithelial paracrin ...... ce during influenza infection.

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Macrophage-epithelial paracrin ...... ce during influenza infection.

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Macrophage-epithelial paracrin ...... ce during influenza infection.

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Journal of Clinical Investigation

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Macrophage-epithelial paracrin ...... ce during influenza infection.

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Balachandar Selvakumar

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Carole Schmoldt

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Emilia Lecuona

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G R Scott Budinger

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Gökhan M Mutlu

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Jacob I Sznajder

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Juergen Lohmeyer

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Julia Bespalowa

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Konstantin Mayer

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Ludger Fink

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P2860

Role of AMP-activated protein kinase in mechanism of metformin action

Functional role of type I and type II interferons in antiviral defense

Proinflammatory cytokine responses induced by influenza A (H5N1) viruses in primary human alveolar and bronchial epithelial cells

Macrophage-expressed IFN-β contributes to apoptotic alveolar epithelial cell injury in severe influenza virus pneumonia

Influenza virus-induced lung injury: pathogenesis and implications for treatment

Phosphorylation-activity relationships of AMPK and acetyl-CoA carboxylase in muscle

Hospitalized patients with 2009 H1N1 influenza in the United States, April-June 2009

Fatal outcome of human influenza A (H5N1) is associated with high viral load and hypercytokinemia

Upregulation of alveolar epithelial active Na+ transport is dependent on beta2-adrenergic receptor signaling

Severe reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2.

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1566-1580

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10.1172/JCI83931

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4

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126

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István Vadász

Christin Peteranderl

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2016-03-21T00:00:00Z

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26999599

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