Salmonella disrupts lymph node architecture by TLR4-mediated suppression of homeostatic chemokines.
about
Immune response and immunopathology during toxoplasmosisThe regulation of T follicular helper responses during infectionNodeomics: pathogen detection in vertebrate lymph nodes using meta-transcriptomics.Mass spectrometry imaging identifies palmitoylcarnitine as an immunological mediator during Salmonella Typhimurium infection.Lymphoadenopathy during lyme borreliosis is caused by spirochete migration-induced specific B cell activationDisrupted lymph node and splenic stroma in mice with induced inflammatory melanomas is associated with impaired recruitment of T and dendritic cells.Mast cell mediator responses and their suppression by pathogenic and commensal microorganisms.Salmonella typhimurium impedes innate immunity with a mast-cell-suppressing protein tyrosine phosphatase, SptP.Innate immune activation during Salmonella infection initiates extramedullary erythropoiesis and splenomegalyS1P-Dependent trafficking of intracellular yersinia pestis through lymph nodes establishes Buboes and systemic infection.Potential immunosuppressive effects of Escherichia coli O157:H7 experimental infection on the bovine host.Tumor-induced stromal reprogramming drives lymph node transformation.MyD88- and TRIF-independent induction of type I interferon drives naive B cell accumulation but not loss of lymph node architecture in Lyme disease.Lymphatics: at the interface of immunity, tolerance, and tumor metastasis.Innate immune control of Salmonella enterica serovar Typhimurium: mechanisms contributing to combating systemic Salmonella infection.SOCS3 revisited: a broad regulator of disease, now ready for therapeutic use?Borrelia burgdorferi Manipulates Innate and Adaptive Immunity to Establish Persistence in Rodent Reservoir Hosts.Cathepsin K activity controls injury-related vascular repair in mice.Marginal reticular cells: a stromal subset directly descended from the lymphoid tissue organizer.TLR-mediated loss of CD62L focuses B cell traffic to the spleen during Salmonella typhimurium infection.Infection with Toxoplasma gondii alters lymphotoxin expression associated with changes in splenic architecture.Lipopolysaccharide induces acute bursal atrophy in broiler chicks by activating TLR4-MAPK-NF-κB/AP-1 signaling.A TNF-p100 pathway subverts noncanonical NF-κB signaling in inflamed secondary lymphoid organs.Subversion of innate and adaptive immune activation induced by structurally modified lipopolysaccharide from Salmonella typhimurium.Chikungunya virus impairs draining lymph node function by inhibiting HEV-mediated lymphocyte recruitmentDiminished nuclear RNA decay upon infection upregulates antibacterial noncoding RNAsNecroptosis of infiltrated macrophages drives Yersinia pestis dispersal within buboes
P2860
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P2860
Salmonella disrupts lymph node architecture by TLR4-mediated suppression of homeostatic chemokines.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@ast
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@en
type
label
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@ast
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@en
prefLabel
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@ast
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@en
P2860
P356
P1433
P1476
Salmonella disrupts lymph node ...... ion of homeostatic chemokines.
@en
P2093
Ashley L St John
Soman N Abraham
P2860
P2888
P304
P356
10.1038/NM.2036
P407
P577
2009-10-25T00:00:00Z