The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells
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Critical role of c-Jun overexpression in liver metastasis of human breast cancer xenograft modelInteracting regions in Stat3 and c-Jun that participate in cooperative transcriptional activationDominant negative c-jun inhibits activation of the cyclin D1 and cyclin E kinase complexescJun overexpression in MCF-7 breast cancer cells produces a tumorigenic, invasive and hormone resistant phenotypeIdentification of SWI.SNF complex subunit BAF60a as a determinant of the transactivation potential of Fos/Jun dimersThe oncogene c-Jun impedes somatic cell reprogrammingAtonal homolog 1 is a tumor suppressor geneCell fate determination factor DACH1 inhibits c-Jun-induced contact-independent growthCyclin A is a c-Jun target gene and is necessary for c-Jun-induced anchorage-independent growth in RAT1a cellsAP-1 in cell proliferation and survivalDirect and indirect roles of cyclin-dependent kinase 5 as an upstream regulator in the c-Jun NH2-terminal kinase cascade: relevance to neurotoxic insults in Alzheimer's disease.Regulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion.Down-regulation of particulate protein kinase Cepsilon and up-regulation of nuclear activator protein-1 DNA binding in liver following in vivo exposure of B6C3F1 male mice to heptachlor epoxide.Heparin (GAG-hed) inhibits LCR activity of human papillomavirus type 18 by decreasing AP1 binding.The human papillomavirus type 16 E7 gene product interacts with and trans-activates the AP1 family of transcription factorsInvolvement of Ras/Raf/AP-1 in BMP-4 signaling during Xenopus embryonic development.Direct and indirect interactions between calcineurin-NFAT and MEK1-extracellular signal-regulated kinase 1/2 signaling pathways regulate cardiac gene expression and cellular growth.Leishmania-induced inactivation of the macrophage transcription factor AP-1 is mediated by the parasite metalloprotease GP63.AP1 regulation of proliferation and initiation of apoptosis in erythropoietin-dependent erythroid cellsTumor necrosis factor alpha gene regulation: enhancement of C/EBPbeta-induced activation by c-Jun.The Stress-response protein prostate-associated gene 4, interacts with c-Jun and potentiates its transactivation.Rb binds c-Jun and activates transcriptionC/EBPbeta (NF-M) is essential for activation of the p20K lipocalin gene in growth-arrested chicken embryo fibroblasts.The mammalian Jun proteins: redundancy and specificity.Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells.Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinasesShortage of mitogen-activated protein kinase is responsible for resistance to AP-1 transactivation and transformation in mouse JB6 cells.Hepatitis B virus HBx protein induces transcription factor AP-1 by activation of extracellular signal-regulated and c-Jun N-terminal mitogen-activated protein kinasesBlocking activator protein-1 activity, but not activating retinoic acid response element, is required for the antitumor promotion effect of retinoic acidc-Jun activation-dependent tumorigenic transformation induced paradoxically by overexpression or block of S-adenosylmethionine decarboxylase.A Jun-binding protein related to a putative tumor suppressorCellular transformation and malignancy induced by ras require c-jun.Lack of a role for Jun kinase and AP-1 in Fas-induced apoptosis.SPARC and thrombospondin genes are repressed by the c-jun oncogene in rat embryo fibroblasts.The c-Jun-induced transformation process involves complex regulation of tenascin-C expression.A dominant negative form of the transcription factor c-Jun affects genes that have opposing effects on lipid homeostasis in miceThe 2 Faces of JNK Signaling in Cancer.Heterodimer formation of cJun and ATF-2 is responsible for induction of c-jun by the 243 amino acid adenovirus E1A proteinCytokines increase CRE binding but decrease CRE-mediated reporter activity in rat hepatocytes by increasing c-Jun.Radiation-induced acid ceramidase confers prostate cancer resistance and tumor relapse.
P2860
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P2860
The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
The transactivating domain of ...... sformation of rat embryo cells
@ast
The transactivating domain of ...... sformation of rat embryo cells
@en
type
label
The transactivating domain of ...... sformation of rat embryo cells
@ast
The transactivating domain of ...... sformation of rat embryo cells
@en
prefLabel
The transactivating domain of ...... sformation of rat embryo cells
@ast
The transactivating domain of ...... sformation of rat embryo cells
@en
P2093
P2860
P356
P1476
The transactivating domain of ...... sformation of rat embryo cells
@en
P2093
B Binétruy
M J Birrer
R K Rosenberg
P2860
P304
P356
10.1128/MCB.11.12.6286
P407
P577
1991-12-01T00:00:00Z