KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways - Wikidata - wikimedia - TriplyDB

KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways

KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways

http://www.wikidata.org/entity/Q36748426

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Endothelial-Monocyte Activating Polypeptide II Suppresses the In Vitro Glioblastoma-Induced Angiogenesis by Inducing Autophagy.Ibrutinib, a Bruton's tyrosine kinase inhibitor, exhibits antitumoral activity and induces autophagy in glioblastoma.Salvianolic acid B, a novel autophagy inducer, exerts antitumor activity as a single agent in colorectal cancer cells.Renoprotective effects of emodin against diabetic nephropathy in rat models are mediated via PI3K/Akt/GSK-3β and Bax/caspase-3 signaling pathways.The KCa3.1 blocker TRAM34 reverses renal damage in a mouse model of established diabetic nephropathy.Oxidative stress promotes myocardial fibrosis by upregulating KCa3.1 channel expression in AGT-REN double transgenic hypertensive mice.Effect of Qufengtongluo Decoction on PI3K/Akt Signaling Pathway in the Kidney of Type 2 Diabetes Mellitus Rat (GK Rat) with Diabetic Nephropathy.Histone Deacetylases Enhance Ca-Activated K⁺ Channel K3.1 Expression in Murine Inflammatory CD4⁺ T Cells

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P2860

KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways

http://www.wikidata.org/entity/Q36748426

description

2016 nî lūn-bûn

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Scientific Reports

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KCa3.1 mediates dysfunction of ...... 3k/Akt/mTOR signaling pathways

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Carol A Pollock

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Chunling Huang

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Delfine Cheng

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Mike Z Lin

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Xin-Ming Chen

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P2860

Modulators of Small- and Intermediate-Conductance Calcium-Activated Potassium Channels and their Therapeutic Indications

mTOR: from growth signal integration to cancer, diabetes and ageing

Role of nitrosative stress and peroxynitrite in the pathogenesis of diabetic complications. Emerging new therapeutical strategies

Molecular mechanisms of diabetic kidney disease

Regulation of autophagy by TGF-β: emerging role in kidney fibrosis

Autophagy fights disease through cellular self-digestion

Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice

Loss of autophagy in the central nervous system causes neurodegeneration in mice

Up-regulation of the IKCa1 potassium channel during T-cell activation. Molecular mechanism and functional consequences

Blockade of the intermediate-conductance calcium-activated potassium channel as a new therapeutic strategy for restenosis

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10.1038/SREP23884

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