Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue. - Wikidata - wikimedia - TriplyDB

Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

http://www.wikidata.org/entity/Q36755620

about

Hyaluronic acid-coated chitosan nanoparticles induce ROS-mediated tumor cell apoptosis and enhance antitumor efficiency by targeted drug delivery via CD44 Characterization of Cardiovascular Alterations Induced by Different Chronic Cisplatin Treatments.Platinum-induced ototoxicity: a review of prevailing ototoxicity criteria.Chronic adventitial inflammation, vasa vasorum expansion, and 5-lipoxygenase up-regulation in irradiated arteries from cancer survivors.Management of ischemic coronary disease in patients receiving chemotherapy: an uncharted clinical challenge.Comprehensive review on cardio-oncology: Role of multimodality imaging.SCF-KIT signaling induces endothelin-3 synthesis and secretion: Thereby activates and regulates endothelin-B-receptor for generating temporally- and spatially-precise nitric oxide to modulate SCF- and or KIT-expressing cell functions.Incidence, time of occurrence and response to heart failure therapy in patients with anthracycline cardiotoxicity.Cardiac Metabolic Deregulation Induced by the Tyrosine Kinase Receptor Inhibitor Sunitinib is rescued by Endothelin Receptor Antagonism Early Actions of Anti-Vascular Endothelial Growth Factor/Vascular Endothelial Growth Factor Receptor Drugs on Angiogenic Blood Vessels.The ethics of radiation exposure in cancer-treated patients : Editorial for: Frequent MUGA testing in a myeloma patient: a case-based ethics discussion.Enhancement of cisplatin cytotoxicity by Retigeric acid B involves blocking DNA repair and activating DR5 in prostate cancer cells.Protective Mechanism of Hydrogen Sulfide against Chemotherapy-Induced Cardiotoxicity.Cardio-oncology: conflicting priorities of anticancer treatment and cardiovascular outcome.A Prospective Study to Evaluate the Effect of Paclitaxel on Cardiac Ejection Fraction.Cardiovascular Toxicities of Small Molecule Tyrosine Kinase Inhibitors: An Opportunity for Systems-Based Approaches.Cardiotoxicity with vascular endothelial growth factor inhibitor therapy Cardio-oncology: A Focus on Cardiotoxicity

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Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

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2016 nî lūn-bûn

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

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Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

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Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

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Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue.

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CIRCULATIONAHA.115.018347

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Vascular Toxicities of Cancer Therapies: The Old and the New--An Evolving Avenue

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Abdul Hakeem

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Cezar A Iliescu

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Cindy L Grines

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Joerg Herrmann

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Konstantinos Charitakis

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Konstantinos Marmagkiolis

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Konstantinos Toutouzas

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Massoud A Leesar

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Mehmet Cilingiroglu

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P2860

Development of coronary artery stenosis in a patient with metastatic renal cell carcinoma treated with sorafenib

Abl family kinases regulate endothelial barrier function in vitro and in mice

Cardiac endothelium-myocyte interaction: clinical opportunities for new heart failure therapies regardless of ejection fraction

Ponatinib in refractory Philadelphia chromosome-positive leukemias.

Non-Q-wave myocardial infarction associated with bleomycin and etoposide chemotherapy

Arterial thromboembolic events in patients with metastatic carcinoma treated with chemotherapy and bevacizumab

Cardiotoxicity of continuous intravenous infusion of 5-fluorouracil: clinical study, prevention and physiopathology. Apropos of 13 cases

Acute vascular toxicity after combination chemotherapy with cisplatin, vinblastine, and bleomycin for testicular cancer

Rho-kinase inhibitor prevents bleomycin-induced injury in neonatal rats independent of effects on lung inflammation

Abl kinases are required for vascular function, Tie2 expression, and angiopoietin-1-mediated survival

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1272-1289

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10.1161/CIRCULATIONAHA.115.018347

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