Transcription factor Sp3 represses expression of p21CIP¹ via inhibition of productive elongation by RNA polymerase II.
about
Overexpression of histone deacetylases in cancer cells is controlled by interplay of transcription factors and epigenetic modulatorsEnforcing the pause: transcription factor Sp3 limits productive elongation by RNA polymerase II.Induction of p21CIP1 protein and cell cycle arrest after inhibition of Aurora B kinase is attributed to aneuploidy and reactive oxygen species.Genome-wide promoter binding profiling of protein phosphatase-1 and its major nuclear targeting subunits.A role for p38 in transcriptional elongation of p21 (CIP1) in response to Aurora B inhibitionHDAC8 and STAT3 repress BMF gene activity in colon cancer cells.
P2860
Transcription factor Sp3 represses expression of p21CIP¹ via inhibition of productive elongation by RNA polymerase II.
description
2013 nî lūn-bûn
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2013年の論文
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name
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@ast
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@en
type
label
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@ast
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@en
prefLabel
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@ast
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@en
P2093
P2860
P356
P1476
Transcription factor Sp3 repre ...... ongation by RNA polymerase II.
@en
P2093
Alvaro Valin
Grace Gill
Jian Ouyang
P2860
P304
P356
10.1128/MCB.00323-12
P407
P577
2013-02-11T00:00:00Z