Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats. - Wikidata - wikimedia - TriplyDB

Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

http://www.wikidata.org/entity/Q36768258

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Physical exercise-induced protection on ischemic cardiovascular and cerebrovascular diseases Energy intake and exercise as determinants of brain health and vulnerability to injury and disease Exercise attenuates neurological deficits by stimulating a critical HSP70/NF-κB/IL-6/synapsin I axis in traumatic brain injury rats.Sodium tanshinone IIA sulfonate improves inflammation, aortic endothelial cell apoptosis, disseminated intravascular coagulation and multiple organ damage in a rat heat stroke model The impact of dietary energy intake on cognitive aging.Forced exercise attenuates neuropathic pain in chronic constriction injury of male rat: an investigation of oxidative stress and inflammation A forced running wheel system with a microcontroller that provides high-intensity exercise training in an animal ischemic stroke model.Exercise preconditioning protects against spinal cord injury in rats by upregulating neuronal and astroglial heat shock protein 72.Aberrant subcellular neuronal calcium regulation in aging and Alzheimer's disease Impaired adaptive cellular responses to oxidative stress and the pathogenesis of Alzheimer's disease Voluntary Exercise Preconditioning Activates Multiple Antiapoptotic Mechanisms and Improves Neurological Recovery after Experimental Traumatic Brain Injury.Exercise preconditioning exhibits neuroprotective effects on hippocampal CA1 neuronal damage after cerebral ischemia.Cellular and molecular neurobiology of brain preconditioning.Treadmill exercise preconditioning attenuates lung damage caused by systemic endotoxemia in type 1 diabetic rats.Decreasing or increasing heat shock protein 72 exacerbates or attenuates heat-induced cell death, respectively, in rat hypothalamic cells.Exercise pretraining attenuates endotoxin-induced hemodynamic alteration in type I diabetic rats.Beneficial Effects of Exercise Pretreatment in a Sporadic Alzheimer's Rat Model.Mild endotoxemia, NF-kappaB translocation, and cytokine increase during exertional heat stress in trained and untrained individuals.

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

http://www.wikidata.org/entity/Q36768258

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2007 nî lūn-bûn

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

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British Journal of Sports Medicine

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Exercise pretraining protects against cerebral ischaemia induced by heat stroke in rats.

@en

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Ching-Hsia Hung

http://www.w3.org/2001/XMLSchema#string

Mao-Tsun Lin

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Sheng-Hsien Chen

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Willy Chou

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Yi-Ming Lo

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Yu-Wen Chen

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P2860

Morbidity and mortality associated with the July 1980 heat wave in St. Louis and Kansas City, Mo

The Hsp70 and Hsp60 chaperone machines

Molecular chaperones in cellular protein folding

Heat shock protein 72 overexpression protects against hyperthermia, circulatory shock, and cerebral ischemia during heatstroke.

Heat shock proteins and cardiovascular pathophysiology.

Ischemia-reperfusion-related repair deficit after oxidative stress: implications of faulty transcripts in neuronal sensitivity after brain injury.

Cerebrovascular dysfunction is an attractive target for therapy in heat stroke.

Human umbilical cord blood cells or estrogen may be beneficial in treating heatstroke.

Heat shock, stress proteins, chaperones, and proteotoxicity.

Nitric oxide mediates glutamate neurotoxicity in primary cortical cultures.

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9

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cerebral infarction

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