Unfolded protein response in Drosophila: why another model can make it fly.
about
Characterization of the properties of a novel mutation in VAPB in familial amyotrophic lateral sclerosisAdaptive preconditioning in neurological diseases – therapeutic insights from proteostatic perturbationsIntegration of UPRER and oxidative stress signaling in the control of intestinal stem cell proliferationThe retromer complex is required for rhodopsin recycling and its loss leads to photoreceptor degenerationExploring the Conserved Role of MANF in the Unfolded Protein Response in Drosophila melanogasterCharacterization of the structural and functional determinants of MANF/CDNF in Drosophila in vivo modelLoss of p24 function in Drosophila melanogaster causes a stress response and increased levels of NF-kappaB-regulated gene products.Transcriptional regulation by CHIP/LDB complexesThe PERK pathway independently triggers apoptosis and a Rac1/Slpr/JNK/Dilp8 signaling favoring tissue homeostasis in a chronic ER stress Drosophila modelDrosophila XBP1 expression reporter marks cells under endoplasmic reticulum stress and with high protein secretory load.Ire1 mediated mRNA splicing in a C-terminus deletion mutant of Drosophila Xbp1Getting the better of ER stress.PERK Limits Drosophila Lifespan by Promoting Intestinal Stem Cell Proliferation in Response to ER Stress.Serum Amyloid P Component Ameliorates Neurological Damage Caused by Expressing a Lysozyme Variant in the Central Nervous System of Drosophila melanogaster.ER stress inhibits neuronal death by promoting autophagyA Drosophila gain-of-function screen for candidate genes involved in steroid-dependent neuroendocrine cell remodelingLoss of Drosophila i-AAA protease, dYME1L, causes abnormal mitochondria and apoptotic degeneration.A modified UPR stress sensing system reveals a novel tissue distribution of IRE1/XBP1 activity during normal Drosophila development.ER stress protects from retinal degenerationRegulation of the transcriptome by ER stress: non-canonical mechanisms and physiological consequences.Drosophila melanogaster activating transcription factor 4 regulates glycolysis during endoplasmic reticulum stress.Boudin trafficking reveals the dynamic internalisation of specific septate junction components in Drosophila.Hyung Don Ryoo: a healthy career in cellular death.Detrimental effects of proteasome inhibition activity in Drosophila melanogaster: implication of ER stress, autophagy, and apoptosis.
P2860
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P2860
Unfolded protein response in Drosophila: why another model can make it fly.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Unfolded protein response in Drosophila: why another model can make it fly.
@ast
Unfolded protein response in Drosophila: why another model can make it fly.
@en
type
label
Unfolded protein response in Drosophila: why another model can make it fly.
@ast
Unfolded protein response in Drosophila: why another model can make it fly.
@en
prefLabel
Unfolded protein response in Drosophila: why another model can make it fly.
@ast
Unfolded protein response in Drosophila: why another model can make it fly.
@en
P356
P1433
P1476
Unfolded protein response in Drosophila: why another model can make it fly.
@en
P2093
Hyung Don Ryoo
P304
P356
10.4161/CC.6.7.4064
P577
2007-04-23T00:00:00Z