Thiazolidinedione drugs block cardiac KATP channels and may increase propensity for ischaemic ventricular fibrillation in pigs. - Wikidata - wikimedia - TriplyDB

Thiazolidinedione drugs block cardiac KATP channels and may increase propensity for ischaemic ventricular fibrillation in pigs.

Thiazolidinedione drugs block cardiac KATP channels and may increase propensity for ischaemic ventricular fibrillation in pigs.

http://www.wikidata.org/entity/Q36787371

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PPAR-γ as a therapeutic target in cardiovascular disease: evidence and uncertainty Minireview: Challenges and opportunities in development of PPAR agonists Chemical proteomics-based analysis of off-target binding profiles for rosiglitazone and pioglitazone: clues for assessing potential for cardiotoxicity.Mitochondrial KATP channel inhibition blunts arrhythmia protection in ischemic exercised hearts.Cardiac ion channel modulation by the hypoglycaemic agent rosiglitazone Effects of rosiglitazone on the configuration of action potentials and ion currents in canine ventricular cells.Rosiglitazone inhibits Kv4.3 potassium channels by open-channel block and acceleration of closed-state inactivation.Can the electrophysiological action of rosiglitazone explain its cardiac side effects?Rosiglitazone selectively inhibits K(ATP) channels by acting on the K(IR) 6 subunit Thiazolidinedione drugs promote onset, alter characteristics, and increase mortality of ischemic ventricular fibrillation in pigs.PPARγ and Its Role in Cardiovascular Diseases.The potential effects of anti-diabetic medications on myocardial ischemia-reperfusion injury.Cardioprotective effects of incretin during ischaemia-reperfusion.The Positive Regulation of eNOS Signaling by PPAR Agonists in Cardiovascular Diseases.Rosiglitazone inhibits vascular KATP channels and coronary vasodilation produced by isoprenaline.PPARγ activator, rosiglitazone: Is it beneficial or harmful to the cardiovascular system?Anti-arrhythmic effect of diosgenin in reperfusion-induced myocardial injury in a rat model: activation of nitric oxide system and mitochondrial KATP channel.Mechanisms responsible for beneficial and adverse effects of rosiglitazone in a rat model of acute cardiac ischaemia-reperfusion.Pharmacokinetics of pioglitazone after multiple oral dose administration in horses.Metformin prevents ischaemic ventricular fibrillation in metabolically normal pigs.Effect of rosiglitazone on cardiac electrophysiology, infarct size and mitochondrial function in ischaemia and reperfusion of swine and rat heart.Blockade of human HERG K⁺ channels by rosiglitazone, an antidiabetic drug.

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P2860

Thiazolidinedione drugs block cardiac KATP channels and may increase propensity for ischaemic ventricular fibrillation in pigs.

http://www.wikidata.org/entity/Q36787371

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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Thiazolidinedione drugs block ...... tricular fibrillation in pigs.

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Thiazolidinedione drugs block ...... tricular fibrillation in pigs.

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Thiazolidinedione drugs block ...... tricular fibrillation in pigs.

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Thiazolidinedione drugs block ...... tricular fibrillation in pigs.

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C R Greyson

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P2860

Effect of Rosiglitazone on the Risk of Myocardial Infarction and Death from Cardiovascular Causes

Secondary prevention of macrovascular events in patients with type 2 diabetes in the PROactive Study (PROspective pioglitAzone Clinical Trial In macroVascular Events): a randomised controlled trial

Effects of the I(K.ATP) blockers glibenclamide and HMR1883 on cardiac electrophysiology during ischemia and reperfusion.

ATP-sensitive potassium channel blocker HMR 1883 reduces mortality and ischemia-associated electrocardiographic changes in pigs with coronary occlusion.

HMR 1883, a cardioselective K(ATP) channel blocker, inhibits ischaemia- and reperfusion-induced ventricular fibrillation in rats.

Consequences of cardiac myocyte-specific ablation of KATP channels in transgenic mice expressing dominant negative Kir6 subunits.

Pioglitazone and risk of cardiovascular events in patients with type 2 diabetes mellitus: a meta-analysis of randomized trials.

Inhibitory action of insulin-sensitizing agents on calcium channels in smooth muscle cells from resistance arteries of guinea-pig.

KATP channels and 'border zone' arrhythmias: role of the repolarization dispersion between normal and ischaemic ventricular regions.

Sensitivity of Kir6.2-SUR1 currents, in the absence and presence of sodium azide, to the K(ATP) channel inhibitors, ciclazindol and englitazone

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