Systemic LPS induces spinal inflammatory gene expression and impairs phrenic long-term facilitation following acute intermittent hypoxia.
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Unexpected benefits of intermittent hypoxia: enhanced respiratory and nonrespiratory motor functionMicrocircuits in respiratory rhythm generation: commonalities with other rhythm generating networks and evolutionary perspectives.Therapeutic potential of intermittent hypoxia: a matter of dose.Chronic intermittent hypoxia exerts CNS region-specific effects on rat microglial inflammatory and TLR4 gene expression.Intermittent Hypoxia-Induced Spinal Inflammation Impairs Respiratory Motor Plasticity by a Spinal p38 MAP Kinase-Dependent MechanismRespiratory function after selective respiratory motor neuron death from intrapleural CTB-saporin injectionsRepetitive acute intermittent hypoxia does not promote generalized inflammatory gene expression in the rat CNS.Protective effects of intermittent hypoxia on brain and memory in a mouse model of apnea of prematurity.Intermittent hypoxia and neurorehabilitation.Tumor necrosis factor-α potentiates long-term potentiation in the rat dentate gyrus after acute hypoxia.Common mechanisms of compensatory respiratory plasticity in spinal neurological disorders.Neurotoxic saboteurs: straws that break the hippo's (hippocampus) back drive cognitive impairment and Alzheimer's Disease.Spinal metaplasticity in respiratory motor controlThe impact of inflammation on respiratory plasticity.Phrenic motor outputs in response to bronchopulmonary C-fibre activation following chronic cervical spinal cord injury.Neuropathology in respiratory-related motoneurons in young Pompe (Gaa(-/-)) mice.Adenosine-dependent phrenic motor facilitation is inflammation resistant.Cyclooxygenase enzyme activity does not impair respiratory motor plasticity after one night of intermittent hypoxia.Pharmacological modulation of hypoxia-induced respiratory neuroplasticity.Phrenic long-term facilitation following intrapleural CTB-SAP-induced respiratory motor neuron death.Effect of acute intermittent hypoxia on motor function in individuals with chronic spinal cord injury following ibuprofen pretreatment: A pilot study.Systemic Inflammation Inhibits Serotonin Receptor 2-induced Phrenic Motor Facilitation Upstream from BDNF/TrkB Signaling.Systemic Lipopolysaccharide-Induced Pain Sensitivity and Spinal Inflammation Were Reduced by Minocycline in Neonatal Rats
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Systemic LPS induces spinal inflammatory gene expression and impairs phrenic long-term facilitation following acute intermittent hypoxia.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Systemic LPS induces spinal in ...... ng acute intermittent hypoxia.
@en
type
label
Systemic LPS induces spinal in ...... ng acute intermittent hypoxia.
@en
prefLabel
Systemic LPS induces spinal in ...... ng acute intermittent hypoxia.
@en
P2093
P2860
P1476
Systemic LPS induces spinal in ...... ng acute intermittent hypoxia.
@en
P2093
A G Huxtable
G S Mitchell
J J Watters
S M C Smith
P2860
P304
P356
10.1152/JAPPLPHYSIOL.01347.2012
P407
P577
2013-01-17T00:00:00Z