Contribution of NF-kappa B and Sp1 binding motifs to the replicative capacity of human immunodeficiency virus type 1: distinct patterns of viral growth are determined by T-cell types
about
Transcriptional activation of the integrated chromatin-associated human immunodeficiency virus type 1 promoterDifferent members of the Sp1 multigene family exert opposite transcriptional regulation of the long terminal repeat of HIV-1Role of the basic domain of human immunodeficiency virus type 1 matrix in macrophage infectionInhibition of HIV-1 replication in primary human monocytes by the IkappaB-alphaS32/36A repressor of NF-kappaBSingle amino acid changes in the human immunodeficiency virus type 1 matrix protein block virus particle productionCrystal Structure of NFAT Bound to the HIV-1 LTR Tandem κB Enhancer ElementStructural Basis of HIV-1 Activation by NF-κB—A Higher-Order Complex of p50:RelA Bound to the HIV-1 LTRInhibition of human immunodeficiency virus type 1 transcription by chemical cyclin-dependent kinase inhibitorsDivergent transcriptional regulation among expanding human immunodeficiency virus type 1 subtypesAdaptation of HIV-1 depends on the host-cell environment.Control of stochastic gene expression by host factors at the HIV promoter.Regulation of HIV-1 transcription in cells of the monocyte-macrophage lineage.Functional properties of the HIV-1 long terminal repeat containing single-nucleotide polymorphisms in Sp site III and CCAAT/enhancer binding protein site I.Coexpression of NF-kappa B/Rel and Sp1 transcription factors in human immunodeficiency virus 1-induced, dendritic cell-T-cell syncytia.Construction and characterization of a temperature-sensitive human immunodeficiency virus type 1 reverse transcriptase mutantNaturally occurring human immunodeficiency virus type 1 long terminal repeats have a frequently observed duplication that binds RBF-2 and represses transcription.Development of a self-inactivating lentivirus vector.Mechanisms of HIV Transcriptional Regulation by Drugs of Abuse.Genetic variation and HIV-associated neurologic disease.OX40 stimulation by gp34/OX40 ligand enhances productive human immunodeficiency virus type 1 infection.NF-kappaB cis-acting motifs of the human immunodeficiency virus (HIV) long terminal repeat regulate HIV transcription in human macrophages.Combinatorial latency reactivation for HIV-1 subtypes and variantsCritical contacts between HIV-1 integrase and viral DNA identified by structure-based analysis and photo-crosslinking.Identification of c-fos-responsive elements downstream of TAR in the long terminal repeat of human immunodeficiency virus type-1.Chromatin disruption in the promoter of human immunodeficiency virus type 1 during transcriptional activation.A cooperative interaction between NF-kappa B and Sp1 is required for HIV-1 enhancer activation.Regulation of Mycobacterium tuberculosis-dependent HIV-1 transcription reveals a new role for NFAT5 in the toll-like receptor pathway.p6Gag is required for particle production from full-length human immunodeficiency virus type 1 molecular clones expressing proteaseThe tale of the long tail: the cytoplasmic domain of HIV-1 gp41.RelB-p50 NF-kappa B complexes are selectively induced by cytomegalovirus immediate-early protein 1: differential regulation of Bcl-x(L) promoter activity by NF-kappa B family membersThe molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit.Innate and adaptive factors regulating human immunodeficiency virus type 1 genomic activation.Development of co-selected single nucleotide polymorphisms in the viral promoter precedes the onset of human immunodeficiency virus type 1-associated neurocognitive impairment.The cytoplasmic domain of the HIV-1 glycoprotein gp41 induces NF-κB activation through TGF-β-activated kinase 1.Inhibition of human immunodeficiency virus type 1 replication by a Tat-activated, transduced interferon gene: targeted expression to human immunodeficiency virus type 1-infected cellsVirion incorporation of envelope glycoproteins with long but not short cytoplasmic tails is blocked by specific, single amino acid substitutions in the human immunodeficiency virus type 1 matrix.Inhibition of apoptosis in human immunodeficiency virus-infected cells enhances virus production and facilitates persistent infection.Multiple effects of mutations in human immunodeficiency virus type 1 integrase on viral replicationIsolation and characterization of a syncytium-inducing, macrophage/T-cell line-tropic human immunodeficiency virus type 1 isolate that readily infects chimpanzee cells in vitro and in vivoSelective infection of human T-lymphotropic virus type 1 (HTLV-1)-infected cells by chimeric human immunodeficiency viruses containing HTLV-1 tax response elements in the long terminal repeat.
P2860
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P2860
Contribution of NF-kappa B and Sp1 binding motifs to the replicative capacity of human immunodeficiency virus type 1: distinct patterns of viral growth are determined by T-cell types
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Contribution of NF-kappa B and ...... are determined by T-cell types
@en
type
label
Contribution of NF-kappa B and ...... are determined by T-cell types
@en
prefLabel
Contribution of NF-kappa B and ...... are determined by T-cell types
@en
P2093
P2860
P1433
P1476
Contribution of NF-kappa B and ...... are determined by T-cell types
@en
P2093
Buckler-White AJ
P2860
P304
P407
P577
1991-08-01T00:00:00Z