Development of pulmonary fibrosis through a pathway involving the transcription factor Fra-2/AP-1. - Wikidata - wikimedia - TriplyDB

Development of pulmonary fibrosis through a pathway involving the transcription factor Fra-2/AP-1.

Development of pulmonary fibrosis through a pathway involving the transcription factor Fra-2/AP-1.

http://www.wikidata.org/entity/Q36802614

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Suppressor of cytokine signaling (SOCS)-1 is expressed in human prostate cancer and exerts growth-inhibitory function through down-regulation of cyclins and cyclin-dependent kinases Scleroderma pathogenesis: a pivotal role for fibroblasts as effector cells Selective genomic targeting by FRA-2/FOSL2 transcription factor: regulation of the Rgs4 gene is mediated by a variant activator protein 1 (AP-1) promoter sequence/CREB-binding protein (CBP) mechanism Small molecule inhibitors targeting activator protein 1 (AP-1).Transcriptome profiles of carcinoma-in-situ and invasive non-small cell lung cancer as revealed by SAGE Fra-1/AP-1 transcription factor negatively regulates pulmonary fibrosis in vivo.Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis.Fra-2/AP-1 controls bone formation by regulating osteoblast differentiation and collagen production.Loss of JUNB/AP-1 promotes invasive prostate cancer Regulatory T cell-deficient scurfy mice develop systemic autoimmune features resembling lupus-like disease.Progesterone receptor transcriptome and cistrome in decidualized human endometrial stromal cells.Transcription factor ets-2 plays an important role in the pathogenesis of pulmonary fibrosis.FOSL2 promotes leptin gene expression in human and mouse adipocytes.Longitudinal in vivo microcomputed tomography of mouse lungs: No evidence for radiotoxicity.LOXL4 is induced by transforming growth factor β1 through Smad and JunB/Fra2 and contributes to vascular matrix remodeling.The transcription factor c-JUN/AP-1 promotes HBV-related liver tumorigenesis in mice Mechanosensing by the α6-integrin confers an invasive fibroblast phenotype and mediates lung fibrosis.Emerging concepts in the pathogenesis of lung fibrosis Fra-2/AP-1 controls adipocyte differentiation and survival by regulating PPARγ and hypoxia.Pulmonary Hypertension Associated with Idiopathic Pulmonary Fibrosis: Current and Future Perspectives.Characterization of key transcription factors as molecular signatures of HPV-positive and HPV-negative oral cancers.Cellular and molecular aspects of vascular dysfunction in systemic sclerosis.Angiogenesis and vasculopathy in systemic sclerosis: evolving concepts.A process-based review of mouse models of pulmonary hypertension.Animal models of scleroderma: current state and recent development.Double heterozygous mice for Klf5 and Fli1 genes: a new animal model of systemic sclerosis recapitulating its three cardinal pathological features.Fibrogenesis, novel lessons from animal models.MicroRNA 155-deficiency leads to decreased autoantibody levels and reduced severity of nephritis and pneumonitis in pristane-induced lupus.Critical Appraisal of the Utility and Limitations of Animal Models of Scleroderma.Recent advances in animal models of systemic sclerosis.Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genes.Unifying mechanism for different fibrotic diseases.Tamoxifen Inhibits TGF-β-Mediated Activation of Myofibroblasts by Blocking Non-Smad Signaling Through ERK1/2.Meprin β contributes to collagen deposition in lung fibrosis.TGFβ signaling and the control of myofibroblast differentiation: Implications for chronic inflammatory disorders.Pathogenesis of systemic sclerosis-current concept and emerging treatments.New insights into adipocyte-specific leptin gene expression.One time nose-only inhalation of MWCNTs: Exploring the mechanism of toxicity by intermittent sacrifice in Wistar rats.Distinct severe acute respiratory syndrome coronavirus-induced acute lung injury pathways in two different nonhuman primate species.c-Jun N-terminal kinase 1 promotes transforming growth factor-β1-induced epithelial-to-mesenchymal transition via control of linker phosphorylation and transcriptional activity of Smad3.

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Development of pulmonary fibrosis through a pathway involving the transcription factor Fra-2/AP-1.

http://www.wikidata.org/entity/Q36802614

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Proceedings of the National Academy of Sciences of the United States of America

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Development of pulmonary fibro ...... anscription factor Fra-2/AP-1.

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Helmut Popper

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Maria-Helena Idarraga

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Martina Rath

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Peter Hasselblatt

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Rainer Zenz

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Vukoslav Komnenovic

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P2860

The myofibroblast: one function, multiple origins

Up-regulation and profibrotic role of osteopontin in human idiopathic pulmonary fibrosis

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

IL-13 signaling through the IL-13alpha2 receptor is involved in induction of TGF-beta1 production and fibrosis

Osteopontin transcription in aortic vascular smooth muscle cells is controlled by glucose-regulated upstream stimulatory factor and activator protein-1 activities

Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix

AP-1: a double-edged sword in tumorigenesis

The role of CXC chemokines in pulmonary fibrosis.

Overexpression of DeltaFosB transcription factor(s) increases bone formation and inhibits adipogenesis.

Does current knowledge explain the pathogenesis of idiopathic pulmonary fibrosis? A perspective.

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pulmonary fibrosis

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