The level of origin firing inversely affects the rate of replication fork progression.
about
Quantitative BrdU immunoprecipitation method demonstrates that Fkh1 and Fkh2 are rate-limiting activators of replication origins that reprogram replication timing in G1 phase.The Eukaryotic Mismatch Recognition Complexes Track with the Replisome during DNA SynthesisHuman Adipose-Derived Stem Cells Expanded Under Ambient Oxygen Concentration Accumulate Oxidative DNA Lesions and Experience Procarcinogenic DNA Replication StressThe role of Dbf4-dependent protein kinase in DNA polymerase ζ-dependent mutagenesis in Saccharomyces cerevisiae.SIRT7 promotes genome integrity and modulates non-homologous end joining DNA repair.Rif1 regulates initiation timing of late replication origins throughout the S. cerevisiae genome.Replication stress is a potent driver of functional decline in ageing haematopoietic stem cellsBRPF3-HBO1 regulates replication origin activation and histone H3K14 acetylationRtt107 Is a Multi-functional Scaffold Supporting Replication Progression with Partner SUMO and Ubiquitin Ligases.Budding Yeast Rif1 Controls Genome Integrity by Inhibiting rDNA ReplicationConcerted activities of Mcm4, Sld3, and Dbf4 in control of origin activation and DNA replication fork progressionHigh levels of RAD51 perturb DNA replication elongation and cause unscheduled origin firing due to impaired CHK1 activation.Phosphorylation of CMG helicase and Tof1 is required for programmed fork arrest.The Rtt107 BRCT scaffold and its partner modification enzymes collaborate to promote replication.Possible role of H1 histone in replication timing.Human Ribonuclease H1 resolves R loops and thereby enables progression of the DNA replication fork.Mcm10 deficiency causes defective-replisome-induced mutagenesis and a dependency on error-free postreplicative repair.The cohesin acetyltransferase Eco1 coordinates rDNA replication and transcription.MTBP, the partner of Treslin, contains a novel DNA-binding domain that is essential for proper initiation of DNA replication.Fewer forks make faster progress.p53 gain-of-function mutations increase Cdc7-dependent replication initiation.Shortage of dNTPs underlies altered replication dynamics and DNA breakage in the absence of the APC/C cofactor Cdh1.Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.Chromatin conformation regulates the coordination between DNA replication and transcription.
P2860
Q27934621-D5169018-6A90-408C-BEA9-A0B1D8552DB8Q28551589-91CB1128-F87A-4FC2-A55D-D7FF2A229D3FQ33722201-42C3299B-6365-401F-ACA1-28A79D7FB40AQ34016548-7595E079-F2DC-48D2-9771-046A6C94905DQ34528261-170A465D-4B4E-4DE4-8832-4E8E30620A4DQ35176804-9290346A-C8AC-4A1E-93EA-476F013A4B7CQ35684586-3ED788D4-286D-4B33-8BFB-A6ECFDC4FEAAQ35856245-E0BE1CB2-D49C-486B-B936-98FD683591DFQ36172725-74E57874-09D6-4A9A-9A49-FEC0909E50C7Q36184338-CF69D926-0D17-4AAF-8C9B-9B1AF4566321Q36633188-68332811-D2B1-48AD-8D88-42FBC1043A8FQ36780751-4FD49AA1-878A-4E34-9FCF-FAC6DEB3460CQ37065309-685DEDE3-6DF8-4B7A-9930-CB5DF4937BAEQ37289716-35600813-6746-4A76-8404-8375FF5380E0Q38286216-D9D0A19C-A477-49B6-BCE7-C1F7C665DFC8Q38674820-23CC39AB-834C-4412-A91A-8CC425B9FBC4Q42248736-4E2D99D2-33D7-4DAF-86F1-DB67933010FDQ42962562-CDE289C5-32C0-42E8-A60B-7DDAC05A2FBCQ45955691-C15CB759-A8E0-4E3F-A977-204CCC6426DDQ46178570-3C14EF37-75E1-4685-8722-6569018DA7D9Q48100871-D1AE8736-2C89-4137-8889-28A4BAC70661Q50949245-16577DBC-8124-461B-AA0E-1B7C611D5772Q52321974-75D18CE4-6ADE-4C20-879F-A83FF1B991D3Q52717242-5C18B0DA-3492-4969-9D31-D02BC71AF847
P2860
The level of origin firing inversely affects the rate of replication fork progression.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
The level of origin firing inversely affects the rate of replication fork progression.
@ast
The level of origin firing inversely affects the rate of replication fork progression.
@en
type
label
The level of origin firing inversely affects the rate of replication fork progression.
@ast
The level of origin firing inversely affects the rate of replication fork progression.
@en
prefLabel
The level of origin firing inversely affects the rate of replication fork progression.
@ast
The level of origin firing inversely affects the rate of replication fork progression.
@en
P2093
P2860
P356
P1476
The level of origin firing inversely affects the rate of replication fork progression
@en
P2093
Janis M Yee
Jared M Peace
Jeffrey M Jancuska
Marianne Tecklenburg
Sandra K Villwock
Sanket Rege
Simon R V Knott
Simon Tavaré
Tittu Nellimoottil
Yuan Zhong
P2860
P304
P356
10.1083/JCB.201208060
P407
P577
2013-04-01T00:00:00Z