Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma. - Wikidata - wikimedia - TriplyDB

Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma.

Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma.

http://www.wikidata.org/entity/Q36810987

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Antiviral potential of cathelicidins The impact of viral genotype on pathogenesis and disease severity: respiratory syncytial virus and human rhinoviruses Regulatory T Cells Control Immune Responses through Their Non-Redundant Tissue Specific Features.Relationship of Allergy with Asthma: There Are More Than the Allergy "Eggs" in the Asthma "Basket".CD8+ TCR transgenic strains expressing public versus private TCR targeting the respiratory syncytial virus K(d)M2(82-90) epitope demonstrate similar functional profiles.Early-life viral infections and the development of asthma: a target for asthma prevention?Asthma: NHLBI Workshop on the Primary Prevention of Chronic Lung Diseases Immunity to RSV in Early-Life.Antiviral B cell and T cell immunity in the lungs.Neonatal mucosal immunology.Protective and dysregulated T cell immunity in RSV infection The human cathelicidin LL-37 has antiviral activity against respiratory syncytial virus.Toward primary prevention of asthma. Reviewing the evidence for early-life respiratory viral infections as modifiable risk factors to prevent childhood asthma A case for antibiotic perturbation of the microbiota leading to allergy development.Objectives, design and enrollment results from the Infant Susceptibility to Pulmonary Infections and Asthma Following RSV Exposure Study (INSPIRE).Immunostimulatory Defective Viral Genomes from Respiratory Syncytial Virus Promote a Strong Innate Antiviral Response during Infection in Mice and Humans.Cutting Edge: Dual Function of PPARγ in CD11c+ Cells Ensures Immune Tolerance in the Airways Evolution of the Immune Response to Chronic Airway Colonization with Aspergillus fumigatus Hyphae Genes associated with RSV lower respiratory tract infection and asthma: the application of genetic epidemiological methods to understand causality.The Immune Fulcrum: Regulatory T Cells Tip the Balance Between Pro- and Anti-inflammatory Outcomes upon Infection Cathelicidins Have Direct Antiviral Activity against Respiratory Syncytial Virus In Vitro and Protective Function In Vivo in Mice and Humans.Perinatal antibiotic treatment affects murine microbiota, immune responses and allergic asthma Antisense oligonucleotide treatment enhances the recovery of acute lung injury through IL-10-secreting M2-like macrophage-induced expansion of CD4+ regulatory T cells.Virus/allergen interactions in asthma.Risk of childhood asthma following infant bronchiolitis during the respiratory syncytial virus season.Respiratory syncytial virus infection modifies and accelerates pulmonary disease via DC activation and migration.Mitochondrial H2O2 in Lung Antigen-Presenting Cells Blocks NF-κB Activation to Prevent Unwarranted Immune Activation Characterization of Epitope-Specific Anti-Respiratory Syncytial Virus (Anti-RSV) Antibody Responses after Natural Infection and after Vaccination with Formalin-Inactivated RSV T-cell immunoglobulin and mucin domain 1 deficiency eliminates airway hyperreactivity triggered by the recognition of airway cell death.Role of regulatory T cells during virus infection.Cellular and humoral immunity of virus-induced asthma The respiratory syncytial virus fusion protein and neutrophils mediate the airway mucin response to pathogenic respiratory syncytial virus infection.The Advance of Personalized and Stratified Therapies in Bronchial Asthma: Phenotypes - Endotypes - Biomarkers.Cytokine production and signaling pathways in respiratory virus infection.An asthma-associated IL4R variant exacerbates airway inflammation by promoting conversion of regulatory T cells to TH17-like cells Regulatory T cells prevent Th2 immune responses and pulmonary eosinophilia during respiratory syncytial virus infection in mice.Respiratory infections in preterm infants and subsequent asthma: a cohort study.Recent advances in diagnosis, prevention, and treatment of human respiratory syncytial virus.Fallen angels or risen apes? A tale of the intricate complexities of imbalanced immune responses in the pathogenesis and progression of immune-mediated and viral cancers.A brave new world: the lung microbiota in an era of change.

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P2860

Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma.

http://www.wikidata.org/entity/Q36810987

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Early infection with respirato ...... eptibility to allergic asthma.

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Early infection with respirato ...... eptibility to allergic asthma.

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Early infection with respirato ...... eptibility to allergic asthma.

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Early infection with respirato ...... eptibility to allergic asthma.

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Early infection with respirato ...... eptibility to allergic asthma.

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Anupriya Khare

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Anuradha Ray

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Christina Morse

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Mahesh Raundhal

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Manohar Yarlagadda

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Martin L Moore

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Nandini Krishnamoorthy

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Prabir Ray

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R Stokes Peebles

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Sally E Wenzel

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P2860

Evidence of a causal role of winter virus infection during infancy in early childhood asthma

Activation of c-Kit in dendritic cells regulates T helper cell differentiation and allergic asthma

Indoleamine 2,3-dioxygenase in lung dendritic cells promotes Th2 responses and allergic inflammation

The transcription factor GATA-3 is necessary and sufficient for Th2 cytokine gene expression in CD4 T cells

CD127 expression inversely correlates with FoxP3 and suppressive function of human CD4+ T reg cells

Reciprocal TH17 and regulatory T cell differentiation mediated by retinoic acid

GATA3-driven Th2 responses inhibit TGF-beta1-induced FOXP3 expression and the formation of regulatory T cells.

Decrease of Foxp3+ Treg cell number and acquisition of effector cell phenotype during lethal infection

Th2 cells and GATA-3 in asthma: new insights into the regulation of airway inflammation

Priming with secreted glycoprotein G of respiratory syncytial virus (RSV) augments interleukin-5 production and tissue eosinophilia after RSV challenge

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