Subacute inhalation exposure to ozone induces systemic inflammation but not insulin resistance in a diabetic mouse model.
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Repeated ozone exposure exacerbates insulin resistance and activates innate immune response in genetically susceptible miceChemokine (C-C Motif) Receptor-Like 2 is not essential for lung injury, lung inflammation, or airway hyperresponsiveness induced by acute exposure to ozone.Serum amyloid A: an ozone-induced circulating factor with potentially important functions in the lung-brain axis.
P2860
Subacute inhalation exposure to ozone induces systemic inflammation but not insulin resistance in a diabetic mouse model.
description
2016 nî lūn-bûn
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2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
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2016年论文
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2016年论文
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name
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@ast
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@en
type
label
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@ast
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@en
prefLabel
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@ast
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@en
P2093
P2860
P1476
Subacute inhalation exposure t ...... nce in a diabetic mouse model.
@en
P2093
Jack R Harkema
James G Wagner
Katryn Allen
Keisha M Williams
Minjie Chen
Qinghua Sun
Ryan Lewandowski
Sanjay Rajagopalan
Zhekang Ying
P2860
P304
P356
10.3109/08958378.2016.1146808
P50
P577
2016-03-01T00:00:00Z