Analysis of the molecular pathogenesis of cardiomyopathy-causing cTnT mutants I79N, ΔE96, and ΔK210.
about
Genotype-specific pathogenic effects in human dilated cardiomyopathy.A study of tropomyosin's role in cardiac function and disease using thin-filament reconstituted myocardium.Using baculovirus/insect cell expressed recombinant actin to study the molecular pathogenesis of HCM caused by actin mutation A331PPathogenesis associated with a restrictive cardiomyopathy mutant in cardiac troponin T is due to reduced protein stability and greatly increased myofilament Ca2+ sensitivity.
P2860
Analysis of the molecular pathogenesis of cardiomyopathy-causing cTnT mutants I79N, ΔE96, and ΔK210.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
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2013年论文
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name
Analysis of the molecular path ...... mutants I79N, ΔE96, and ΔK210.
@en
type
label
Analysis of the molecular path ...... mutants I79N, ΔE96, and ΔK210.
@en
prefLabel
Analysis of the molecular path ...... mutants I79N, ΔE96, and ΔK210.
@en
P2860
P1433
P1476
Analysis of the molecular path ...... mutants I79N, ΔE96, and ΔK210
@en
P2093
Hannah M Caster
P2860
P304
P356
10.1016/J.BPJ.2013.04.001
P407
P577
2013-05-01T00:00:00Z