Human cytomegalovirus induces a biphasic inflammatory response in primary endothelial cells.

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Human Cytomegalovirus Up-Regulates Endothelin Receptor Type B: Implication for Vasculopathies?Restimulating interest in cytomegalovirus as a cofactor for HIV infection.

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Human cytomegalovirus induces a biphasic inflammatory response in primary endothelial cells.

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http://www.wikidata.org/entity/Q36827140

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2013 nî lūn-bûn

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Human cytomegalovirus induces ...... in primary endothelial cells.

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Human cytomegalovirus induces ...... in primary endothelial cells.

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Human cytomegalovirus induces ...... in primary endothelial cells.

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Human cytomegalovirus induces ...... in primary endothelial cells.

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C Söderberg-Naucler

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H C Jeffery

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L M Butler

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P2860

ICAM-1 regulates neutrophil adhesion and transcellular migration of TNF-alpha-activated vascular endothelium under flow

Monocyte chemoattractant protein 1 acts as a T-lymphocyte chemoattractant

Getting to the site of inflammation: the leukocyte adhesion cascade updated

Molecular mechanisms of leukocyte recruitment in the inflammatory process.

Omega-3 Fatty acids and inflammation: novel interactions reveal a new step in neutrophil recruitment

Human cytomegalovirus inhibits transcription of the CC chemokine MCP-1 gene.

Cytomegalovirus in inflammatory bowel disease: pathogen or innocent bystander?

The role of interleukin-8 in inflammation and mechanisms of regulation.

IL-6 in human cytomegalovirus secretome promotes angiogenesis and survival of endothelial cells through the stimulation of survivin.

Kaposi's sarcoma-associated herpesvirus infection of endothelial cells inhibits neutrophil recruitment through an interleukin-6-dependent mechanism: a new paradigm for viral immune evasion

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6530-6535

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scholarly article

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10.1128/JVI.00265-13

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English

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2013-03-27T00:00:00Z

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236088132

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P698

23536673

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endothelium

Cytomegalovirus

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3648090

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Human cytomegalovirus induces a biphasic inflammatory response in primary endothelial cells.

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P2860

P2860

Human cytomegalovirus induces a biphasic inflammatory response in primary endothelial cells.

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