Change in host cell tropism associated with in vitro replication of equine infectious anemia virus.
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Disease induction by virus derived from molecular clones of equine infectious anemia virus.Biological characterization of Rev variation in equine infectious anemia virusEquine endothelial cells support productive infection of equine infectious anemia virus.Development and characterization of an in vivo pathogenic molecular clone of equine infectious anemia virus.Characterization of a cytolytic strain of equine infectious anemia virus.Characterization of functional domains of equine infectious anemia virus Rev suggests a bipartite RNA-binding domainMonocyte maturation controls expression of equine infectious anemia virus.Analysis of multiple mRNAs from pathogenic equine infectious anemia virus (EIAV) in an acutely infected horse reveals a novel protein, Ttm, derived from the carboxy terminus of the EIAV transmembrane protein.Isolation and characterization of new wild-type isolates of bovine lentivirus.Identification of a hypervariable region in the long terminal repeat of equine infectious anemia virus.Characterization of early pathogenic effects after experimental infection of calves with bovine immunodeficiency-like virus.The surface envelope protein gene region of equine infectious anemia virus is not an important determinant of tropism in vitro.Equine infectious anemia virus derived from a molecular clone persistently infects horses.Inhibition of lentivirus replication by aqueous extracts of Prunella vulgarisLimited sequence heterogeneity among biologically distinct human immunodeficiency virus type 1 isolates from individuals involved in a clustered infectious outbreak.Equine infectious anemia virus entry occurs through clathrin-mediated endocytosis.Evolution of the equine infectious anemia virus long terminal repeat during the alteration of cell tropism.Endocytosis and a low-pH step are required for productive entry of equine infectious anemia virus.Localized sequence heterogeneity in the long terminal repeats of in vivo isolates of equine infectious anemia virus.An equine infectious anemia virus variant superinfects cells through novel receptor interactions.In vivo replicative status and envelope heterogeneity of equine infectious anemia virus in an inapparent carrier.Identification of transactivation-response sequences in the long terminal repeat of bovine immunodeficiency-like virus.Characterization of variable regions in the envelope and S3 open reading frame of equine infectious anemia virus.Subpopulations of equine infectious anemia virus Rev coexist in vivo and differ in phenotype.Characterization of Equine Infectious Anemia Virus Long Terminal Repeat Quasispecies In Vitro and In Vivo.
P2860
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P2860
Change in host cell tropism associated with in vitro replication of equine infectious anemia virus.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Change in host cell tropism as ...... quine infectious anemia virus.
@en
type
label
Change in host cell tropism as ...... quine infectious anemia virus.
@en
prefLabel
Change in host cell tropism as ...... quine infectious anemia virus.
@en
P2860
P1433
P1476
Change in host cell tropism as ...... quine infectious anemia virus.
@en
P2093
Carpenter S
Chesebro B
P2860
P304
P407
P577
1989-06-01T00:00:00Z