Multiple EBNA1-binding sites are required to form an EBNA1-dependent enhancer and to activate a minimal replicative origin within oriP of Epstein-Barr virus.
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EBP2, a human protein that interacts with sequences of the Epstein-Barr virus nuclear antigen 1 important for plasmid maintenance.High-level and high-throughput recombinant protein production by transient transfection of suspension-growing human 293-EBNA1 cellsExpression of Epstein-Barr virus nuclear antigen-1 induces B cell neoplasia in transgenic miceEBNA-1, a bifunctional transcriptional activatorEpstein-Barr virus latent genesEpstein-Barr virus nuclear antigen 1 does not cause lymphoma in C57BL/6J miceHuman RPA (hSSB) interacts with EBNA1, the latent origin binding protein of Epstein-Barr virusEpstein-Barr virus latency switch in human B-cells: a physico-chemical model.Zinc coordination is required for and regulates transcription activation by Epstein-Barr nuclear antigen 1Transcription profiling of Epstein-Barr virus nuclear antigen (EBNA)-1 expressing cells suggests targeting of chromatin remodeling complexesChromatin organization of gammaherpesvirus latent genomes.Epstein-Barr virus nuclear antigen 1 does not induce lymphoma in transgenic FVB mice.Cellular transcription factors enhance herpes simplex virus type 1 oriS-dependent DNA replication.Functional analyses of the EBNA1 origin DNA binding protein of Epstein-Barr virus.The minimal replicator of Epstein-Barr virus oriP.Establishment of latent Epstein-Barr virus infection and stable episomal maintenance in murine B-cell lines.BHRF1 of Epstein-Barr virus, which is homologous to human proto-oncogene bcl2, is not essential for transformation of B cells or for virus replication in vitro.A nuclear matrix attachment region organizes the Epstein-Barr viral plasmid in Raji cells into a single DNA domain.Epstein-Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growthA human TATA binding protein-related protein with altered DNA binding specificity inhibits transcription from multiple promoters and activators.EBV latency types adopt alternative chromatin conformations.Identification of properties of the Kaposi's sarcoma-associated herpesvirus latent origin of replication that are essential for the efficient establishment and maintenance of intact plasmids.Constitutive binding of EBNA1 protein to the Epstein-Barr virus replication origin, oriP, with distortion of DNA structure during latent infection.The spacing between adjacent binding sites in the family of repeats affects the functions of Epstein-Barr nuclear antigen 1 in transcription activation and stable plasmid maintenanceHuman origin recognition complex binds to the region of the latent origin of DNA replication of Epstein-Barr virus.Maintenance of Epstein-Barr virus (EBV) oriP-based episomes requires EBV-encoded nuclear antigen-1 chromosome-binding domains, which can be replaced by high-mobility group-I or histone H1.Separation of the DNA replication, segregation, and transcriptional activation functions of Epstein-Barr nuclear antigen 1.Identification of spliced gammaherpesvirus 68 LANA and v-cyclin transcripts and analysis of their expression in vivo during latent infectionKaposi's Sarcoma-Associated Herpesvirus Genome Replication, Partitioning, and Maintenance in Latency.Phosphoacceptor site S173 in the regulatory domain of Epstein-Barr Virus ZEBRA protein is required for lytic DNA replication but not for activation of viral early genes.Cooperative assembly of EBNA1 on the Epstein-Barr virus latent origin of replicationThe variable 3' ends of a human cytomegalovirus oriLyt transcript (SRT) overlap an essential, conserved replicator element.A papillomavirus E2 phosphorylation mutant exhibits normal transient replication and transcription but is defective in transformation and plasmid retentionBinding of the human papillomavirus E1 origin-recognition protein is regulated through complex formation with the E2 enhancer-binding protein.The bovine papillomavirus origin of replication requires a binding site for the E2 transcriptional activator.Analysis of viral cis elements conferring Kaposi's sarcoma-associated herpesvirus episome partitioning and maintenance.The gammaherpesvirus 68 latency-associated nuclear antigen homolog is critical for the establishment of splenic latency.Retention of plasmid DNA in mammalian cells is enhanced by binding of the Epstein-Barr virus replication protein EBNA1.The EBNA-2 arginine-glycine domain is critical but not essential for B-lymphocyte growth transformation; the rest of region 3 lacks essential interactive domains.Effect of number and position of EBNA-1 binding sites in Epstein-Barr virus oriP on the sites of initiation, barrier formation, and termination of replication
P2860
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P2860
Multiple EBNA1-binding sites are required to form an EBNA1-dependent enhancer and to activate a minimal replicative origin within oriP of Epstein-Barr virus.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Multiple EBNA1-binding sites a ...... in oriP of Epstein-Barr virus.
@en
type
label
Multiple EBNA1-binding sites a ...... in oriP of Epstein-Barr virus.
@en
prefLabel
Multiple EBNA1-binding sites a ...... in oriP of Epstein-Barr virus.
@en
P2860
P1433
P1476
Multiple EBNA1-binding sites a ...... hin oriP of Epstein-Barr virus
@en
P2093
Wysokenski DA
P2860
P304
P407
P577
1989-06-01T00:00:00Z