The spliced BZLF1 gene of Epstein-Barr virus (EBV) transactivates an early EBV promoter and induces the virus productive cycle.
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Epstein-barr virus immediate-early protein BZLF1 is SUMO-1 modified and disrupts promyelocytic leukemia bodiesThe B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1Reactive oxygen species mediate Epstein-Barr virus reactivation by N-methyl-N'-nitro-N-nitrosoguanidineIdentification of Novel Small Organic Compounds with Diverse Structures for the Induction of Epstein-Barr Virus (EBV) Lytic Cycle in EBV-Positive Epithelial MalignanciesThe cellular oncogene c-myb can interact synergistically with the Epstein-Barr virus BZLF1 transactivator in lymphoid cells.Tpl2/AP-1 enhances murine gammaherpesvirus 68 lytic replication.Degradation of phosphorylated p53 by viral protein-ECS E3 ligase complex.CpG-methylation regulates a class of Epstein-Barr virus promoters.Viral response to chemotherapy in endemic burkitt lymphomaThe Epstein-Barr virus lytic transactivator Zta interacts with the helicase-primase replication proteins.Epstein-Barr virus immediate-early proteins BZLF1 and BRLF1 activate the ATF2 transcription factor by increasing the levels of phosphorylated p38 and c-Jun N-terminal kinasesThe Epstein-Barr virus protein BRLF1 activates S phase entry through E2F1 induction.Sumoylation of the Epstein-Barr virus BZLF1 protein inhibits its transcriptional activity and is regulated by the virus-encoded protein kinase.Epstein-Barr virus immediate-early protein BRLF1 induces the lytic form of viral replication through a mechanism involving phosphatidylinositol-3 kinase activation.Epstein-Barr virus immediate-early protein BRLF1 interacts with CBP, promoting enhanced BRLF1 transactivation.BZLF1 activation of the methylated form of the BRLF1 immediate-early promoter is regulated by BZLF1 residue 186.The Epstein-Barr virus R transactivator (Rta) contains a complex, potent activation domain with properties different from those of VP16.Evolutionary aspects of oncogenic herpesvirusesEpstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1.The Epstein-Barr virus BZLF1 protein inhibits tumor necrosis factor receptor 1 expression through effects on cellular C/EBP proteinsNucleoprotein structure of immediate-early promoters Zp and Rp and of oriLyt of latent Epstein-Barr virus genomesThe Epstein-Barr virus immediate-early protein BZLF1 induces both a G(2) and a mitotic blockEpstein-Barr virus mRNA export factor EB2 is essential for production of infectious virus.Use of adenovirus vectors expressing Epstein-Barr virus (EBV) immediate-early protein BZLF1 or BRLF1 to treat EBV-positive tumors.The Epstein-Barr virus immediate-early protein BZLF1 regulates p53 function through multiple mechanismsProtein kinase C-independent activation of the Epstein-Barr virus lytic cycle.Sequence analysis of the Epstein-Barr virus (EBV) BRLF1 gene in nasopharyngeal and gastric carcinomas.Virus and cell RNAs expressed during Epstein-Barr virus replicationLatent membrane protein 1 inhibits Epstein-Barr virus lytic cycle induction and progress via different mechanisms.Epstein-Barr virus Zta upregulates matrix metalloproteinases 3 and 9 that synergistically promote cell invasion in vitro.Epstein-Barr viruses that express a CD21 antibody provide evidence that gp350's functions extend beyond B-cell surface binding.Tegument protein control of latent herpesvirus establishment and animation.Physical and functional interaction of the Epstein-Barr virus BZLF1 transactivator with the retinoic acid receptors RAR alpha and RXR alpha.Direct BRLF1 binding is required for cooperative BZLF1/BRLF1 activation of the Epstein-Barr virus early promoter, BMRF1.Amino acids in the basic domain of Epstein-Barr virus ZEBRA protein play distinct roles in DNA binding, activation of early lytic gene expression, and promotion of viral DNA replication.The Epstein-Barr virus BRRF1 protein, Na, induces lytic infection in a TRAF2- and p53-dependent mannerCellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latencyDifferentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial CellsDetection of heterogeneous Epstein-Barr virus gene expression patterns within individual post-transplantation lymphoproliferative disorders.The Epstein-Barr Virus Lytic Protein BZLF1 as a Candidate Target Antigen for Vaccine Development.
P2860
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P2860
The spliced BZLF1 gene of Epstein-Barr virus (EBV) transactivates an early EBV promoter and induces the virus productive cycle.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
The spliced BZLF1 gene of Epst ...... es the virus productive cycle.
@en
type
label
The spliced BZLF1 gene of Epst ...... es the virus productive cycle.
@en
prefLabel
The spliced BZLF1 gene of Epst ...... es the virus productive cycle.
@en
P2093
P2860
P1433
P1476
The spliced BZLF1 gene of Epst ...... ces the virus productive cycle
@en
P2093
P2860
P304
P407
P577
1989-07-01T00:00:00Z