Expression of H-ras correlates with metastatic potential: evidence for direct regulation of the metastatic phenotype in 10T1/2 and NIH 3T3 cells
about
Complete nucleotide and deduced amino acid sequences of human and murine preprocathepsin L. An abundant transcript induced by transformation of fibroblastsHyaluronan and the hyaluronan receptor RHAMM promote focal adhesion turnover and transient tyrosine kinase activityEndo180 binds to the C-terminal region of type I collagenOrganization of chromatin in cancer cells: role of signalling pathways.Phase I study of QLNC120, a novel EGFR and HER2 kinase inhibitor, in pre-treated patients with HER2-overexpressing advanced breast cancer.Autocrine induction of tumor protease production and invasion by a metallothionein-regulated TGF-beta 1 (Ser223, 225).Amplification and rearrangement of the Kirsten ras oncogene in virus-transformed BALB/c 3T3 cells during malignant tumor progression.Defining a novel cis element in the 3'-untranslated region of mammalian ribonucleotide reductase component R2 mRNA: role in transforming growth factor-beta 1 induced mRNA stabilizationActive erk regulates microtubule stability in H-ras-transformed cells.Molecular determinants of metastatic transformation.Ribonucleotide reductase R2 component is a novel malignancy determinant that cooperates with activated oncogenes to determine transformation and malignant potential.Complementation of two related tumour cell classes during experimental metastasis tagged with different histochemical marker genes.Prognostic significance of ras/p21 alterations in human ovarian cancer.Regulation of collagen I gene expression by rasThe R1 component of mammalian ribonucleotide reductase has malignancy-suppressing activity as demonstrated by gene transfer experiments.Genetic factors and suppression of metastatic ability of v-Ha-ras-transfected rat mammary cancer cells.Quantitative variations in gene expression: possible role in cellular diversification and tumor progression.Gene expression, cellular diversification and tumor progression to the metastatic phenotype.Mutant p53 tumor suppressor alleles release ras-induced cell cycle growth arrest.Persistence of Ha-ras-induced metastatic potential of SP1 mouse mammary tumors despite loss of the Ha-ras shuttle vector.A derivative of chrysin suppresses two-stage skin carcinogenesis by inhibiting mitogen- and stress-activated kinase 1.Histone H3 phosphorylation, immediate-early gene expression, and the nucleosomal response: a historical perspective.Cell-matrix interactions during tumor invasion.An overview of tumor biology.Defining a novel cis-element in the 3'-untranslated region of mammalian ribonucleotide reductase component R2 mRNA. cis-trans-interactions and message stability.Transforming growth factor-beta1 stimulates multiple protein interactions at a unique cis-element in the 3'-untranslated region of the hyaluronan receptor RHAMM mRNA.Prognostic significance of the expression of ras oncogene product in non-small cell lung cancer.Cytoskeletal stiffness, friction, and fluidity of cancer cell lines with different metastatic potential.Gene products which play a role in cancer invasion and metastasis.Role of MSK1 in the malignant phenotype of Ras-transformed mouse fibroblasts.Increased genomic instability and altered chromosomal protein phosphorylation timing in HRAS-transformed mouse fibroblasts.Growth factor-mediated altered expression and regulation of S-adenosylmethionine decarboxylase in a H-ras transformed cell line capable of malignant progression.Histone H1b phosphorylation is dependent upon ongoing transcription and replication in normal and ras-transformed mouse fibroblasts.Basic fibroblast growth factor selectively regulates ornithine decarboxylase gene expression in malignant H-ras transformed cells.The in vivo clearance of Ha-ras transformants by natural killer cells.Fibroblasts transformed by combinations of ras, myc and mutant p53 exhibit increased phosphorylation of histone H1 that is independent of metastatic potential.Increased phosphorylation of histone H1 in mouse fibroblasts transformed with oncogenes or constitutively active mitogen-activated protein kinase kinase.A radiation-induced murine ovarian granulosa cell tumor line: introduction of v-ras gene potentiates a high metastatic ability.Malignant transformation by H-ras results in aberrant regulation of ribonucleotide reductase gene expression by transforming growth factor-beta 1.Identification of a novel heparin binding domain in RHAMM and evidence that it modifies HA mediated locomotion of ras-transformed cells.
P2860
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P2860
Expression of H-ras correlates with metastatic potential: evidence for direct regulation of the metastatic phenotype in 10T1/2 and NIH 3T3 cells
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Expression of H-ras correlates ...... pe in 10T1/2 and NIH 3T3 cells
@en
type
label
Expression of H-ras correlates ...... pe in 10T1/2 and NIH 3T3 cells
@en
prefLabel
Expression of H-ras correlates ...... pe in 10T1/2 and NIH 3T3 cells
@en
P2093
P2860
P356
P1476
Expression of H-ras correlates ...... pe in 10T1/2 and NIH 3T3 cells
@en
P2093
Greenberg AH
McClarty GA
P2860
P304
P356
10.1128/MCB.7.2.830
P407
P577
1987-02-01T00:00:00Z