Pathogenesis of axonal and neuronal damage in multiple sclerosis.
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MR spectroscopy indicates diffuse multiple sclerosis activity during remissionNeuroprotective Effects Against POCD by Photobiomodulation: Evidence from Assembly/Disassembly of the CytoskeletonGap junctions and hemichannels composed of connexins: potential therapeutic targets for neurodegenerative diseasesRole of microglial m1/m2 polarization in relapse and remission of psychiatric disorders and diseasesMyelin gene regulatory factor is required for maintenance of myelin and mature oligodendrocyte identity in the adult CNSNuclear Receptor NR1H3 in Familial Multiple SclerosisHDAC1 nuclear export induced by pathological conditions is essential for the onset of axonal damageAmelioration of experimental autoimmune encephalomyelitis in C57BL/6 mice by photobiomodulation induced by 670 nm lightThe impact of utilizing different optical coherence tomography devices for clinical purposes and in multiple sclerosis trialsGenetic variation in the KIF1B locus influences susceptibility to multiple sclerosisImaging learning: the search for a memory trace.TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.What went wrong? The flawed concept of cerebrospinal venous insufficiency.Rostrocaudal analysis of corpus callosum demyelination and axon damage across disease stages refines diffusion tensor imaging correlations with pathological features.Human Adipose Stromal/Stem Cells from Obese Donors Show Reduced Efficacy in Halting Disease Progression in the Experimental Autoimmune Encephalomyelitis Model of Multiple Sclerosis.Alternative splicing and transcriptome profiling of experimental autoimmune encephalomyelitis using genome-wide exon arrays.Imaging as an Outcome Measure in Multiple Sclerosis.Progressive multiple sclerosis: from pathogenic mechanisms to treatment.The encephalopathy of prematurity--brain injury and impaired brain development inextricably intertwined.Axonal Membranes and Their Domains: Assembly and Function of the Axon Initial Segment and Node of Ranvier.Apolipoprotein E mediation of neuro-inflammation in a murine model of multiple sclerosis.Global N-acetylaspartate declines even in benign multiple sclerosisLipocalin-2 protein deficiency ameliorates experimental autoimmune encephalomyelitis: the pathogenic role of lipocalin-2 in the central nervous system and peripheral lymphoid tissues.Influence of Circulating Endothelin-1 and Asymmetric Dimethylarginine on Whole Brain Circulation Time in Multiple Sclerosis.Changes of statistical structural fluctuations unveils an early compacted degraded stage of PNS myelin.Neuronal I kappa B kinase beta protects mice from autoimmune encephalomyelitis by mediating neuroprotective and immunosuppressive effects in the central nervous system.Energy failure in multiple sclerosis and its investigation using MR techniques.Pathogenic implications of iron accumulation in multiple sclerosis.Review: Mitochondria and disease progression in multiple sclerosisMicroglial nodules in early multiple sclerosis white matter are associated with degenerating axons.Comparative gene expression analysis in mouse models for multiple sclerosis, Alzheimer's disease and stroke for identifying commonly regulated and disease-specific gene changesAssociation between change in normal appearing white matter metabolites and intrathecal inflammation in natalizumab-treated multiple sclerosisHippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.EphA4 receptor tyrosine kinase is a modulator of onset and disease severity of experimental autoimmune encephalomyelitis (EAE)Potential triggers of MSPhotobiomodulation induced by 670 nm light ameliorates MOG35-55 induced EAE in female C57BL/6 mice: a role for remediation of nitrosative stress.Oligodendrocyte fate after spinal cord injury.Nogo-receptor 1 deficiency has no influence on immune cell repertoire or function during experimental autoimmune encephalomyelitis.Body fluid biomarkers in multiple sclerosis: how far we have come and how they could affect the clinic now and in the futureEffect of treatment with interferon beta-1a on changes in voxel-wise magnetization transfer ratio in normal appearing brain tissue and lesions of patients with relapsing-remitting multiple sclerosis: a 24-week, controlled pilot study.
P2860
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P2860
Pathogenesis of axonal and neuronal damage in multiple sclerosis.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Pathogenesis of axonal and neuronal damage in multiple sclerosis.
@en
type
label
Pathogenesis of axonal and neuronal damage in multiple sclerosis.
@en
prefLabel
Pathogenesis of axonal and neuronal damage in multiple sclerosis.
@en
P1433
P1476
Pathogenesis of axonal and neuronal damage in multiple sclerosis.
@en
P2093
Bruce D Trapp
Ranjan Dutta
P304
S22-31; discussion S43-54
P356
10.1212/01.WNL.0000275229.13012.32
P407
P433
22 Suppl 3
P577
2007-05-01T00:00:00Z