Mitochondrial matrix metalloproteinase activation decreases myocyte contractility in hyperhomocysteinemia
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Gelatinase B/MMP-9 in Tumour Pathogenesis and ProgressionCardiac matrix: a clue for future therapyEpigenetic silencing of TIMP4 in heart failure.Electrical stimulation of cardiomyocytes activates mitochondrial matrix metalloproteinase causing electrical remodeling.Oxidative stress and mitochondrial dysfunction across broad-ranging pathologies: toward mitochondria-targeted clinical strategies.MicroRNAs are involved in homocysteine-induced cardiac remodeling.Role of microRNA29b in blood-brain barrier dysfunction during hyperhomocysteinemia: an epigenetic mechanism.Role of matrix metalloproteinase-9 in the development of diabetic retinopathy and its regulation by H-Ras.MMP-9 gene ablation and TIMP-4 mitigate PAR-1-mediated cardiomyocyte dysfunction: a plausible role of dicer and miRNA.Cardiac specific deletion of N-methyl-d-aspartate receptor 1 ameliorates mtMMP-9 mediated autophagy/mitophagy in hyperhomocysteinemia.Folic acid mitigated cardiac dysfunction by normalizing the levels of tissue inhibitor of metalloproteinase and homocysteine-metabolizing enzymes postmyocardial infarction in miceA glimpse of matrix metalloproteinases in diabetic nephropathy.Loss of interstitial collagen causes structural and functional alterations of cardiomyocyte subsarcolemmal mitochondria in acute volume overload.Hydrogen Sulfide Epigenetically Attenuates Homocysteine-Induced Mitochondrial Toxicity Mediated Through NMDA Receptor in Mouse Brain Endothelial (bEnd3) Cells.Abrogation of MMP-9 gene protects against the development of retinopathy in diabetic mice by preventing mitochondrial damage.Effects of mild hyperhomocysteinemia on electron transport chain complexes, oxidative stress, and protein expression in rat cardiac mitochondria.Autophagy mechanism of right ventricular remodeling in murine model of pulmonary artery constriction.Ablation of Matrix Metalloproteinase-9 Prevents Cardiomyocytes Contractile Dysfunction in DiabeticsRestoration of contractility in hyperhomocysteinemia by cardiac-specific deletion of NMDA-R1Matrix metalloproteinases in diabetic retinopathy: potential role of MMP-9.The NO/ONOO-cycle as the central cause of heart failure.Interrelationship between activation of matrix metalloproteinases and mitochondrial dysfunction in the development of diabetic retinopathy.Intracellular regulation of matrix metalloproteinase-2 activity: new strategies in treatment and protection of heart subjected to oxidative stressMatrix metalloproteinase-2 and myocardial oxidative stress injury: beyond the matrix.Intracellular substrate cleavage: a novel dimension in the biochemistry, biology and pathology of matrix metalloproteinases.Autophagy and heart failure: a possible role for homocysteine.Mitochondrial mitophagic mechanisms of myocardial matrix metabolism and remodelling.Vitamin B12 protects against superoxide-induced cell injury in human aortic endothelial cells.The potential of targeting NMDA receptors outside the CNS.MODULATION OF N-METHYL-D-ASPARTATE RECEPTORS IN ISOLATED RAT HEART.MMP-2 is localized to the mitochondria-associated membrane of the heart.Homocysteine induces COX-2 expression in macrophages through ROS generated by NMDA receptor-calcium signaling pathways.Cardiac tissue inhibitor of matrix metalloprotease 4 dictates cardiomyocyte contractility and differentiation of embryonic stem cells into cardiomyocytes: Road to therapy.The effects of the modulation of NMDA receptors by homocysteine thiolactone and dizocilpine on cardiodynamics and oxidative stress in isolated rat heart.Selective homocysteine-lowering gene transfer attenuates pressure overload-induced cardiomyopathy via reduced oxidative stress.Association of hyperhomocysteinemia with left ventricular dilatation and mass in human heart.Effects of DL-homocysteine thiolactone on cardiac contractility, coronary flow, and oxidative stress markers in the isolated rat heart: the role of different gasotransmitters.The effects of homocysteine-related compounds on cardiac contractility, coronary flow, and oxidative stress markers in isolated rat heart.Luteolin Enhances Sarcoplasmic Reticulum Ca2+-ATPase Activity through p38 MAPK Signaling thus Improving Rat Cardiac Function after Ischemia/Reperfusion.
P2860
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P2860
Mitochondrial matrix metalloproteinase activation decreases myocyte contractility in hyperhomocysteinemia
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Mitochondrial matrix metallopr ...... tility in hyperhomocysteinemia
@en
type
label
Mitochondrial matrix metallopr ...... tility in hyperhomocysteinemia
@en
prefLabel
Mitochondrial matrix metallopr ...... tility in hyperhomocysteinemia
@en
P2093
P2860
P1476
Mitochondrial matrix metallopr ...... tility in hyperhomocysteinemia
@en
P2093
Iluiana E Frank
Jayesh Rai
Karni S Moshal
Mahavir Singh
Michael T Tseng
Munish Kumar
Naira Metreveli
Neetu Tyagi
Phani K Patibandla
Srinivas M Tipparaju
P2860
P304
P356
10.1152/AJPHEART.00099.2008
P577
2008-06-20T00:00:00Z