Atherosclerosis following renal injury is ameliorated by pioglitazone and losartan via macrophage phenotype.
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Extracellular vesicles as mediators of vascular inflammation in kidney diseasePPARγ and the Innate Immune System Mediate the Resolution of InflammationIndoxyl Sulfate Promotes Macrophage IL-1β Production by Activating Aryl Hydrocarbon Receptor/NF-κ/MAPK Cascades, but the NLRP3 inflammasome Was Not Activated.Pioglitazone alleviates inflammation in diabetic mice fed a high-fat diet via inhibiting advanced glycation end-product-induced classical macrophage activation.Cucumis sativus Aqueous Fraction Inhibits Angiotensin II-Induced Inflammation and Oxidative Stress In Vitro.Assessment of the Relationship between Carotid Intima-Media Thickening and Early-Stage Diabetic Kidney Disease Coupled with Helicobacter pylori Infection.
P2860
Atherosclerosis following renal injury is ameliorated by pioglitazone and losartan via macrophage phenotype.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Atherosclerosis following rena ...... rtan via macrophage phenotype.
@en
type
label
Atherosclerosis following rena ...... rtan via macrophage phenotype.
@en
prefLabel
Atherosclerosis following rena ...... rtan via macrophage phenotype.
@en
P2093
P2860
P1433
P1476
Atherosclerosis following rena ...... artan via macrophage phenotype
@en
P2093
Ichiei Narita
MacRae F Linton
Patricia G Yancey
Sergio Fazio
Suguru Yamamoto
Valentina Kon
P2860
P356
10.1016/J.ATHEROSCLEROSIS.2015.06.055
P577
2015-07-04T00:00:00Z