Continuous engagement of a self-specific activation receptor induces NK cell tolerance. - Wikidata - wikimedia - TriplyDB

Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

http://www.wikidata.org/entity/Q36851896

about

Activation receptor-induced tolerance of mature NK cells in vivo requires signaling through the receptor and is reversible NK Cell-Mediated Antibody-Dependent Cellular Cytotoxicity in Cancer Immunotherapy Improving the outcome of leukemia by natural killer cell-based immunotherapeutic strategies Natural killer cell tolerance: control by self or self-control?Current perspectives on natural killer cell education and tolerance: emerging roles for inhibitory receptors Innate or adaptive immunity? The example of natural killer cells Expression of the inhibitory receptor NKG2A correlates with increased liver and splenic NK cell response to activating receptor engagement.Expression of CD226 is associated to but not required for NK cell education.Intact NKG2D-independent function of NK cells chronically stimulated with the NKG2D ligand Rae-1 Adaptations of Natural Killer Cells to Self-MHC Class I.Tuning of natural killer cell reactivity by NKp46 and Helios calibrates T cell responses.Re-educating natural killer cells.Mature natural killer cells with phenotypic and functional alterations accumulate upon sustained stimulation with IL-15/IL-15Ralpha complexes.The requirement for NKG2D in NK cell-mediated rejection of parental bone marrow grafts is determined by MHC class I expressed by the graft recipient.Balancing natural killer cell activation through paired receptors.Natural killer cell dependent within-host competition arises during multiple MCMV infection: consequences for viral transmission and evolution.The discontinuity theory of immunity Homeostatic proliferation generates long-lived natural killer cells that respond against viral infection.Natural killer cells from children with type 1 diabetes have defects in NKG2D-dependent function and signaling IL-2-dependent tuning of NK cell sensitivity for target cells is controlled by regulatory T cells.Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains.NK cells and immune "memory"Acquisition of activation receptor ligand by trogocytosis renders NK cells hyporesponsive.HLA and KIR genotyping correlates with relapse after T-cell-replete haploidentical transplantation in chronic myeloid leukaemia patients.Dual Requirement of Cytokine and Activation Receptor Triggering for Cytotoxic Control of Murine Cytomegalovirus by NK Cells Ionomycin Treatment Renders NK Cells Hyporesponsive.Activation by SLAM Family Receptors Contributes to NK Cell Mediated "Missing-Self" Recognition.Prenatal Allospecific NK Cell Tolerance Hinges on Instructive Allorecognition through the Activating Receptor during Development.NK cell tolerance of self-specific activating receptor KIR2DS1 in individuals with cognate HLA-C2 ligand Tolerance of NK cells encountering their viral ligand during development.Characterization of a novel NKG2D and NKp46 double-mutant mouse reveals subtle variations in the NK cell repertoire.Natural killer cells and their receptors in multiple sclerosis.HLA-C-dependent prevention of leukemia relapse by donor activating KIR2DS1.Natural killer cell licensing in mice with inducible expression of MHC class I.Activating receptors promote NK cell expansion for maintenance, IL-10 production, and CD8 T cell regulation during viral infection.Cognate HLA absence in trans diminishes human NK cell education.Controlling natural killer cell responses: integration of signals for activation and inhibition.Contrasting roles of the IL-1 and IL-18 receptors in MyD88-dependent contact hypersensitivity.Natural killer cells remember: an evolutionary bridge between innate and adaptive immunity?

P2860

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P2860

Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

http://www.wikidata.org/entity/Q36851896

description

2008 nî lūn-bûn

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2008年の論文

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2008年论文

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2008年论文

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2008年论文

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name

Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

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Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

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Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

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P1433

Journal of Experimental Medicine

P1476

Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

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P2093

Achim Schneeberger

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Jeanette T Pingel

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Liping Yang

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Peter A Keyel

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Sandeep K Tripathy

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Tammy P Cheng

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Wayne M Yokoyama

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P2860

The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor

Toll-like receptors 9 and 3 as essential components of innate immune defense against mouse cytomegalovirus infection

Structural elucidation of the m157 mouse cytomegalovirus ligand for Ly49 natural killer cell receptors

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

NKG2D recruits two distinct adapters to trigger NK cell activation and costimulation

Selective associations with signaling proteins determine stimulatory versus costimulatory activity of NKG2D

In search of the 'missing self': MHC molecules and NK cell recognition

Retinoic acid early inducible genes define a ligand family for the activating NKG2D receptor in mice

Tolerance and alloreactivity of the Ly49D subset of murine NK cells.

Recognition of a virus-encoded ligand by a natural killer cell activation receptor.

P304

1829-1841

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10.1084/JEM.20072446

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8

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205

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5245872

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18606857

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2525593

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