Gene silencing in adipose tissue macrophages regulates whole-body metabolism in obese mice. - Wikidata - wikimedia - TriplyDB

Gene silencing in adipose tissue macrophages regulates whole-body metabolism in obese mice.

Gene silencing in adipose tissue macrophages regulates whole-body metabolism in obese mice.

http://www.wikidata.org/entity/Q36855257

about

Emerging evidence for beneficial macrophage functions in atherosclerosis and obesity-induced insulin resistance Metabolic functions of FABPs--mechanisms and therapeutic implications Perinatal overnutrition exacerbates adipose tissue inflammation caused by high-fat feeding in C57BL/6J mice Osteopontin deletion prevents the development of obesity and hepatic steatosis via impaired adipose tissue matrix remodeling and reduced inflammation and fibrosis in adipose tissue and liver in mice Adipose extracellular matrix remodelling in obesity and insulin resistance Endotrophin triggers adipose tissue fibrosis and metabolic dysfunction Lipid storage by adipose tissue macrophages regulates systemic glucose tolerance Macrophage recruitment in obese adipose tissue TGR5 reduces macrophage migration through mTOR-induced C/EBPβ differential translation IL-1 signaling in obesity-induced hepatic lipogenesis and steatosis Biomaterial-mediated modification of the local inflammatory environment.Activated Kupffer cells inhibit insulin sensitivity in obese mice.A major role of insulin in promoting obesity-associated adipose tissue inflammation.Elastin Insufficiency Predisposes Mice to Impaired Glucose Metabolism.The Roles of Adipokines, Proinflammatory Cytokines, and Adipose Tissue Macrophages in Obesity-Associated Insulin Resistance in Modest Obesity and Early Metabolic Dysfunction.Tenomodulin promotes human adipocyte differentiation and beneficial visceral adipose tissue expansion.Silencing CCR2 in Macrophages Alleviates Adipose Tissue Inflammation and the Associated Metabolic Syndrome in Dietary Obese Mice.Bone marrow leptin signaling mediates obesity-associated adipose tissue inflammation in male mice.Peptide- and Amine-Modified Glucan Particles for the Delivery of Therapeutic siRNA.Morphological and inflammatory changes in visceral adipose tissue during obesity.Heme oxygenase-1: an emerging therapeutic target to curb cardiac pathology.Liver innate immune cells and insulin resistance: the multiple facets of Kupffer cells.Lipid signaling and lipotoxicity in metaflammation: indications for metabolic disease pathogenesis and treatment.Ubiquitous transgenic overexpression of C-C chemokine ligand 2: a model to assess the combined effect of high energy intake and continuous low-grade inflammation.Local proliferation of macrophages contributes to obesity-associated adipose tissue inflammation Hyperplasia, de novo lymphangiogenesis, and lymphatic regression in mice with tissue-specific, inducible overexpression of murine VEGF-D.Assessment of the number and function of macrophages in the placenta of gestational diabetes mellitus patients.The inflammatory status score including IL-6, TNF-α, osteopontin, fractalkine, MCP-1 and adiponectin underlies whole-body insulin resistance and hyperglycemia in type 2 diabetes mellitus.Decreasing CB1 receptor signaling in Kupffer cells improves insulin sensitivity in obese mice.Sucrose Non-Fermenting Related Kinase Regulates Both Adipose Inflammation and Energy Homeostasis in Mice and Humans.Attenuated Low-Grade Inflammation Following Long-Term Dietary Intervention in Postmenopausal Women with Obesity.Hepatocyte-secreted DPP4 in obesity promotes adipose inflammation and insulin resistance.Macrophage Lamin A/C Regulates Inflammation and the Development of Obesity-Induced Insulin Resistance.Macrophage-Targeted Therapeutics for Metabolic Disease.Pu'erh tea extract-mediated protection against hepatosteatosis and insulin resistance in mice with diet-induced obesity is associated with the induction of de novo lipogenesis in visceral adipose tissue.CRISPR-delivery particles targeting nuclear receptor-interacting protein 1 () in adipose cells to enhance energy expenditure

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P2860

Gene silencing in adipose tissue macrophages regulates whole-body metabolism in obese mice.

http://www.wikidata.org/entity/Q36855257

description

2013 nî lūn-bûn

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2013年の論文

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2013年论文

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2013年论文

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name

Gene silencing in adipose tiss ...... body metabolism in obese mice.

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Gene silencing in adipose tiss ...... body metabolism in obese mice.

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Gene silencing in adipose tiss ...... body metabolism in obese mice.

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PNAS.1300492110

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Gene silencing in adipose tiss ...... body metabolism in obese mice.

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P2093

Jessica L Cohen

http://www.w3.org/2001/XMLSchema#string

Joseph C Yawe

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Michael P Czech

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Michaela Tencerova

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Myriam Aouadi

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Pranitha Vangala

http://www.w3.org/2001/XMLSchema#string

Sarah M Nicoloro

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Shinya U Amano

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P2860

Obesity is associated with macrophage accumulation in adipose tissue

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

Reversal of obesity- and diet-induced insulin resistance with salicylates or targeted disruption of Ikkbeta

Increased adipose tissue expression of tumor necrosis factor-alpha in human obesity and insulin resistance

Macrophages, inflammation, and insulin resistance

Interleukin-1-receptor antagonist in type 2 diabetes mellitus

Mechanisms of TNF-alpha-induced insulin resistance.

Adipocyte dysfunctions linking obesity to insulin resistance and type 2 diabetes.

Overexpression of monocyte chemoattractant protein-1 in adipose tissues causes macrophage recruitment and insulin resistance.

Fibroblast growth factor 21-deficient mice demonstrate impaired adaptation to ketosis.

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