Latency of Epstein-Barr virus is disrupted by gain-of-function mutant cellular AP-1 proteins that preferentially bind methylated DNA.
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Regulation of the latent-lytic switch in Epstein-Barr virusEpstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesisMethyl-dependent and spatial-specific DNA recognition by the orthologous transcription factors human AP-1 and Epstein-Barr virus Zta5-hydroxymethylation of the EBV genome regulates the latent to lytic switch.ERK/c-Jun Recruits Tet1 to Induce Zta Expression and Epstein-Barr Virus Reactivation through DNA DemethylationMethylation of Epstein-Barr virus Rta promoter in EBV primary infection, reactivation and lymphoproliferation.Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes.Epstein-Barr virus encoded microRNAs target SUMO-regulated cellular functions.
P2860
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P2860
Latency of Epstein-Barr virus is disrupted by gain-of-function mutant cellular AP-1 proteins that preferentially bind methylated DNA.
description
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name
Latency of Epstein-Barr virus ...... rentially bind methylated DNA.
@en
type
label
Latency of Epstein-Barr virus ...... rentially bind methylated DNA.
@en
prefLabel
Latency of Epstein-Barr virus ...... rentially bind methylated DNA.
@en
P2093
P2860
P356
P1476
Latency of Epstein-Barr virus ...... rentially bind methylated DNA.
@en
P2093
George Miller
Kuan-Ping Yu
Lee Heston
Richard Park
Ruth Wang'ondu
Zhaowei Ding
P2860
P304
P356
10.1073/PNAS.1301577110
P407
P577
2013-04-26T00:00:00Z