ELR-negative CXC chemokine CXCL11 (IP-9/I-TAC) facilitates dermal and epidermal maturation during wound repair. - Wikidata - wikimedia - TriplyDB

ELR-negative CXC chemokine CXCL11 (IP-9/I-TAC) facilitates dermal and epidermal maturation during wound repair.

ELR-negative CXC chemokine CXCL11 (IP-9/I-TAC) facilitates dermal and epidermal maturation during wound repair.

http://www.wikidata.org/entity/Q36855716

about

IP-10 induces dissociation of newly formed blood vessels Chemokines in Wound Healing and as Potential Therapeutic Targets for Reducing Cutaneous Scarring The Role of Chemokines in Fibrotic Wound Healing CXCR3 in carcinoma progression The Beginning of the End: CXCR3 Signaling in Late-Stage Wound Healing Innate immune response of human alveolar macrophages during influenza A infection Innate immune response to influenza A virus in differentiated human alveolar type II cells m-Calpain activation is regulated by its membrane localization and by its binding to phosphatidylinositol 4,5-bisphosphate.Can scarring be turned off?Lack of CXC chemokine receptor 3 signaling leads to hypertrophic and hypercellular scarring Delayed reepithelialization and basement membrane regeneration after wounding in mice lacking CXCR3.CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans An IP-10 (CXCL10)-derived peptide inhibits angiogenesis.Skin wound healing and scarring: fetal wounds and regenerative restitution.Delayed cutaneous wound closure in HO-2 deficient mice despite normal HO-1 expression Fibronectin binding modulates CXCL11 activity and facilitates wound healing CXCR3 chemokine receptor enables local CD8(+) T cell migration for the destruction of virus-infected cells.Pericyte regulation of vascular remodeling through the CXC receptor 3 Inflammation and wound healing: the role of the macrophage Matrix control of scarring.Epithelial and mesenchymal phenotypic switchings modulate cell motility in metastasis Targeting tumor cell motility as a strategy against invasion and metastasis.CXCL11 Expression by Keratinocytes Occurs Transiently Between Reaching Confluence and Cellular Compaction.Critical Role of IP-10 on Reducing Experimental Corneal Neovascularization.Altered CXCR3 isoform expression regulates prostate cancer cell migration and invasion.Expression analysis of the early chemokine response 4 h after in vitro traumatic brain injury.Local Probiotic Therapy with Lactobacillus plantarum Mitigates Scar Formation in Rabbits after Burn Injury and Infection.Transplanted fibroblasts prevents dysfunctional repair in a murine CXCR3-deficient scarring model.Multipotent stromal cells/mesenchymal stem cells and fibroblasts combine to minimize skin hypertrophic scarring.Chemokines and Their Receptors Are Key Players in the Orchestra That Regulates Wound Healing.Chemokine Signaling during Midline Epithelial Seam Disintegration Facilitates Palatal Fusion.The Role of CXCR3 and Its Chemokine Ligands in Skin Disease and Cancer

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P2860

ELR-negative CXC chemokine CXCL11 (IP-9/I-TAC) facilitates dermal and epidermal maturation during wound repair.

http://www.wikidata.org/entity/Q36855716

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

2008年论文

@zh

2008年论文

@zh-cn

name

ELR-negative CXC chemokine CXC ...... aturation during wound repair.

@en

type

label

ELR-negative CXC chemokine CXC ...... aturation during wound repair.

@en

prefLabel

ELR-negative CXC chemokine CXC ...... aturation during wound repair.

@en

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AJPATH.2008.070990

P1433

The American Journal of Pathology

P1476

ELR-negative CXC chemokine CXC ...... maturation during wound repair

@en

P2093

Amy Y-Chen

http://www.w3.org/2001/XMLSchema#string

Cecelia C Yates

http://www.w3.org/2001/XMLSchema#string

Diana Whaley

http://www.w3.org/2001/XMLSchema#string

Patricia A Hebda

http://www.w3.org/2001/XMLSchema#string

Priya Kulesekaran

http://www.w3.org/2001/XMLSchema#string

P2860

Human IP-9: A keratinocyte-derived high affinity CXC-chemokine ligand for the IP-10/Mig receptor (CXCR3)

CXC chemokine receptor 3 expression on CD34(+) hematopoietic progenitors from human cord blood induced by granulocyte-macrophage colony-stimulating factor: chemotaxis and adhesion induced by its ligands, interferon gamma-inducible protein 10 and mon

An alternatively spliced variant of CXCR3 mediates the inhibition of endothelial cell growth induced by IP-10, Mig, and I-TAC, and acts as functional receptor for platelet factor 4

IP-10 blocks vascular endothelial growth factor-induced endothelial cell motility and tube formation via inhibition of calpain

Requirement of the chemokine receptor CXCR3 for acute allograft rejection

Delayed and deficient dermal maturation in mice lacking the CXCR3 ELR-negative CXC chemokine receptor

Glu-Leu-Arg-negative CXC chemokine interferon gamma inducible protein-9 as a mediator of epidermal-dermal communication during wound repair

Interferon-inducible protein 9 (CXCL11)-induced cell motility in keratinocytes requires calcium flux-dependent activation of mu-calpain.

Chemokines in cutaneous wound healing.

Activation of m-calpain (calpain II) by epidermal growth factor is limited by protein kinase A phosphorylation of m-calpain

P304

643-652

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scholarly article

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10.2353/AJPATH.2008.070990

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173

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23139966

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18669615

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2527079

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