Brain injury biomarkers are not dependent on β-amyloid in normal elderly.
about
Suspected non-Alzheimer disease pathophysiology--concept and controversyA focus on structural brain imaging in the Alzheimer's disease neuroimaging initiativeEvolving Evidence for the Value of Neuroimaging Methods and Biological Markers in Subjects Categorized with Subjective Cognitive Decline.Diabetes and elevated hemoglobin A1c levels are associated with brain hypometabolism but not amyloid accumulation.Rates of β-amyloid accumulation are independent of hippocampal neurodegeneration.Neuroprotective pathways: lifestyle activity, brain pathology, and cognition in cognitively normal older adults.Biological heterogeneity in ADNI amnestic mild cognitive impairment.Primary age-related tauopathy (PART): a common pathology associated with human agingPART is part of Alzheimer disease.PART, a distinct tauopathy, different from classical sporadic Alzheimer disease.Early detection of Alzheimer's disease using PiB and FDG PETPART and SNAP.Tau aggregation influences cognition and hippocampal atrophy in the absence of beta-amyloid: a clinico-imaging-pathological study of primary age-related tauopathy (PART).Molecular imaging of Alzheimer disease pathologySynergistic effect of β-amyloid and neurodegeneration on cognitive decline in clinically normal individuals.Age-specific population frequencies of cerebral β-amyloidosis and neurodegeneration among people with normal cognitive function aged 50-89 years: a cross-sectional studyMild cognitive impairment with suspected nonamyloid pathology (SNAP): Prediction of progressionDifferential white matter connectivity in early mild cognitive impairment according to CSF biomarkersWhat is normal in normal aging? Effects of aging, amyloid and Alzheimer's disease on the cerebral cortex and the hippocampusSpatially distinct atrophy is linked to β-amyloid and tau in preclinical Alzheimer diseaseBrain structure and function as mediators of the effects of amyloid on memory.Pulse pressure in relation to tau-mediated neurodegeneration, cerebral amyloidosis, and progression to dementia in very old adultsDepressive Symptoms and Biomarkers of Alzheimer's Disease in Cognitively Normal Older Adults.Subtle Cognitive Decline and Biomarker Staging in Preclinical Alzheimer's DiseaseSuspected non-AD pathology in mild cognitive impairmentDifferent definitions of neurodegeneration produce similar amyloid/neurodegeneration biomarker group findingsRole of β-Amyloidosis and Neurodegeneration in Subsequent Imaging Changes in Mild Cognitive ImpairmentTransition rates between amyloid and neurodegeneration biomarker states and to dementia: a population-based, longitudinal cohort studyAssociations between Alzheimer disease biomarkers, neurodegeneration, and cognition in cognitively normal older people.Amyloid imaging in cognitively normal individuals, at-risk populations and preclinical Alzheimer's disease.Hippocampal subfield volumetry in mild cognitive impairment, Alzheimer's disease and semantic dementia.Biomarker-based prediction of progression in MCI: Comparison of AD signature and hippocampal volume with spinal fluid amyloid-β and tauAmyloid-first and neurodegeneration-first profiles characterize incident amyloid PET positivitySelective worsening of brain injury biomarker abnormalities in cognitively normal elderly persons with β-amyloidosis.Hypothetical Preclinical Alzheimer Disease Groups and Longitudinal Cognitive Change.Biomarker modeling of Alzheimer's disease.Alzheimer disease: Aβ-independent processes-rethinking preclinical AD.The Alzheimer's disease mitochondrial cascade hypothesis: progress and perspectives.Amyloid deposition is linked to aberrant entorhinal activity among cognitively normal older adults.Alzheimer disease therapy--moving from amyloid-β to tau.
P2860
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P2860
Brain injury biomarkers are not dependent on β-amyloid in normal elderly.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Brain injury biomarkers are not dependent on β-amyloid in normal elderly.
@en
type
label
Brain injury biomarkers are not dependent on β-amyloid in normal elderly.
@en
prefLabel
Brain injury biomarkers are not dependent on β-amyloid in normal elderly.
@en
P2093
P2860
P50
P356
P1433
P1476
Brain injury biomarkers are not dependent on β-amyloid in normal elderly.
@en
P2093
Bradley F Boeve
Heather J Wiste
Jeffrey L Gunter
Matthew L Senjem
Michelle M Mielke
Prashanthi Vemuri
Rosebud O Roberts
Stephen D Weigand
Val J Lowe
P2860
P304
P356
10.1002/ANA.23816
P577
2013-02-19T00:00:00Z