Brain injury biomarkers are not dependent on β-amyloid in normal elderly. - Wikidata - wikimedia - TriplyDB

Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

http://www.wikidata.org/entity/Q36863815

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Suspected non-Alzheimer disease pathophysiology--concept and controversy A focus on structural brain imaging in the Alzheimer's disease neuroimaging initiative Evolving Evidence for the Value of Neuroimaging Methods and Biological Markers in Subjects Categorized with Subjective Cognitive Decline.Diabetes and elevated hemoglobin A1c levels are associated with brain hypometabolism but not amyloid accumulation.Rates of β-amyloid accumulation are independent of hippocampal neurodegeneration.Neuroprotective pathways: lifestyle activity, brain pathology, and cognition in cognitively normal older adults.Biological heterogeneity in ADNI amnestic mild cognitive impairment.Primary age-related tauopathy (PART): a common pathology associated with human aging PART is part of Alzheimer disease.PART, a distinct tauopathy, different from classical sporadic Alzheimer disease.Early detection of Alzheimer's disease using PiB and FDG PET PART and SNAP.Tau aggregation influences cognition and hippocampal atrophy in the absence of beta-amyloid: a clinico-imaging-pathological study of primary age-related tauopathy (PART).Molecular imaging of Alzheimer disease pathology Synergistic effect of β-amyloid and neurodegeneration on cognitive decline in clinically normal individuals.Age-specific population frequencies of cerebral β-amyloidosis and neurodegeneration among people with normal cognitive function aged 50-89 years: a cross-sectional study Mild cognitive impairment with suspected nonamyloid pathology (SNAP): Prediction of progression Differential white matter connectivity in early mild cognitive impairment according to CSF biomarkers What is normal in normal aging? Effects of aging, amyloid and Alzheimer's disease on the cerebral cortex and the hippocampus Spatially distinct atrophy is linked to β-amyloid and tau in preclinical Alzheimer disease Brain structure and function as mediators of the effects of amyloid on memory.Pulse pressure in relation to tau-mediated neurodegeneration, cerebral amyloidosis, and progression to dementia in very old adults Depressive Symptoms and Biomarkers of Alzheimer's Disease in Cognitively Normal Older Adults.Subtle Cognitive Decline and Biomarker Staging in Preclinical Alzheimer's Disease Suspected non-AD pathology in mild cognitive impairment Different definitions of neurodegeneration produce similar amyloid/neurodegeneration biomarker group findings Role of β-Amyloidosis and Neurodegeneration in Subsequent Imaging Changes in Mild Cognitive Impairment Transition rates between amyloid and neurodegeneration biomarker states and to dementia: a population-based, longitudinal cohort study Associations between Alzheimer disease biomarkers, neurodegeneration, and cognition in cognitively normal older people.Amyloid imaging in cognitively normal individuals, at-risk populations and preclinical Alzheimer's disease.Hippocampal subfield volumetry in mild cognitive impairment, Alzheimer's disease and semantic dementia.Biomarker-based prediction of progression in MCI: Comparison of AD signature and hippocampal volume with spinal fluid amyloid-β and tau Amyloid-first and neurodegeneration-first profiles characterize incident amyloid PET positivity Selective worsening of brain injury biomarker abnormalities in cognitively normal elderly persons with β-amyloidosis.Hypothetical Preclinical Alzheimer Disease Groups and Longitudinal Cognitive Change.Biomarker modeling of Alzheimer's disease.Alzheimer disease: Aβ-independent processes-rethinking preclinical AD.The Alzheimer's disease mitochondrial cascade hypothesis: progress and perspectives.Amyloid deposition is linked to aberrant entorhinal activity among cognitively normal older adults.Alzheimer disease therapy--moving from amyloid-β to tau.

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Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

http://www.wikidata.org/entity/Q36863815

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2013 nî lūn-bûn

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2013年论文

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2013年论文

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name

Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

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type

label

Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

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prefLabel

Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

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P1433

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Annals of Neurology

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Brain injury biomarkers are not dependent on β-amyloid in normal elderly.

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P2093

Bradley F Boeve

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Heather J Wiste

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Jeffrey L Gunter

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Matthew L Senjem

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Michelle M Mielke

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Prashanthi Vemuri

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Rosebud O Roberts

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Stephen D Weigand

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Val J Lowe

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Toward defining the preclinical stages of Alzheimer's disease: recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease

11C PiB and structural MRI provide complementary information in imaging of Alzheimer's disease and amnestic mild cognitive impairment

Automated Anatomical Labeling of Activations in SPM Using a Macroscopic Anatomical Parcellation of the MNI MRI Single-Subject Brain

Voxel-based morphometry--the methods

Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade

Whole brain segmentation: automated labeling of neuroanatomical structures in the human brain

Unified segmentation

Origins of Alzheimer's disease: reconciling cerebrospinal fluid biomarker and neuropathology data regarding the temporal sequence of amyloid-beta and tau involvement.

Measurement of MRI scanner performance with the ADNI phantom.

Cerebrospinal fluid tau and ptau(181) increase with cortical amyloid deposition in cognitively normal individuals: implications for future clinical trials of Alzheimer's disease

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472-480

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scholarly article

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10.1002/ANA.23816

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4

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73

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Ronald C. Petersen

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2013-02-19T00:00:00Z

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235670296

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23424032

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3660408

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