Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.
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The Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and AgingThe Complex Roles of Mechanistic Target of Rapamycin in Adipocytes and Beyond.Novel Browning Agents, Mechanisms, and Therapeutic Potentials of Brown Adipose Tissue.Rapamycin Blocks Induction of the Thermogenic Program in White Adipose Tissue.Raptor/mTORC1 loss in adipocytes causes progressive lipodystrophy and fatty liver disease.The tumor suppressor FLCN mediates an alternate mTOR pathway to regulate browning of adipose tissueReduced ATGL-mediated lipolysis attenuates β-adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue.Mitochondrial homeostasis in adipose tissue remodeling.Ubiquitin Ligases and Posttranslational Regulation of Energy in the Heart: The Hand that Feeds.Beige Adipocyte Maintenance Is Regulated by Autophagy-Induced Mitochondrial Clearance.mTORC1 is Required for Brown Adipose Tissue Recruitment and Metabolic Adaptation to Cold.Characterization of immortalized human brown and white pre-adipocyte cell models from a single donor.SYK kinase mediates brown fat differentiation and activation.The glucagon-like peptide-1 receptor in the ventromedial hypothalamus reduces short-term food intake in male mice by regulating nutrient sensor activity.Natriuretic peptides promote glucose uptake in a cGMP-dependent manner in human adipocytes.Rheb Inhibits Beiging of White Adipose Tissue via PDE4D5-Dependent Downregulation of the cAMP-PKA Signaling Pathway.Cardiac natriuretic peptides promote adipose 'browning' through mTOR complex-1.Membrane Trafficking Protein CDP138 Regulates Fat Browning and Insulin Sensitivity through Controlling Catecholamine Release.Regulation of adiposity by mTORC1.Taming the Flames: Targeting White Adipose Tissue Browning in Hypermetabolic Conditions.Effect of beta-agonists on LAM progression and treatment.GSK3 is a negative regulator of the thermogenic program in brown adipocytes.Response gene to complement 32 suppresses adipose tissue thermogenic genes through inhibiting β3-adrenergic receptor/mTORC1 signaling.Adipose tissue browning: mTOR branches out.Insulin action and resistance in obesity and type 2 diabetes.Adipocyte-specific deletion of Lkb1 and mTOR protects against high-fat diet induced obesity but results in insulin resistance.Mitophagy is required for brown adipose tissue mitochondrial homeostasis during cold challenge.Brown Adipose TissueInhibition of TBK1/IKKε Promotes Regeneration of Pancreatic β-cells
P2860
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P2860
Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.
@en
type
label
Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.
@en
prefLabel
Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.
@en
P2093
P2860
P356
P1476
Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning
@en
P2093
Adilson Guilherme
Chaoying Zhang
Huafeng Fang
Jian-Liang Li
Kalyani Guntur
Marica Bordicchia
Michael P Czech
Sheila Collins
P2860
P304
P356
10.1172/JCI83532
P407
P50
P577
2016-03-28T00:00:00Z