Notch signaling indirectly promotes chondrocyte hypertrophy via regulation of BMP signaling and cell cycle arrest.

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Human Mesenchymal Stromal Cell Sheet Enhances Allograft Repair in a Mouse Model Indirect immobilized Jagged1 suppresses cell cycle progression and induces odonto/osteogenic differentiation in human dental pulp cells.Notch Signaling in Development, Tissue Homeostasis, and Disease.

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Notch signaling indirectly promotes chondrocyte hypertrophy via regulation of BMP signaling and cell cycle arrest.

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http://www.wikidata.org/entity/Q36870956

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

@yue

2016年論文

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2016年論文

@zh-hk

2016年論文

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2016年論文

@zh-tw

2016年论文

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2016年论文

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2016年论文

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name

Notch signaling indirectly pro ...... gnaling and cell cycle arrest.

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type

Item

label

Notch signaling indirectly pro ...... gnaling and cell cycle arrest.

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prefLabel

Notch signaling indirectly pro ...... gnaling and cell cycle arrest.

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P1476

Notch signaling indirectly pro ...... ignaling and cell cycle arrest

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Jinwu Wang

http://www.w3.org/2001/XMLSchema#string

John V Marymont

http://www.w3.org/2001/XMLSchema#string

Massimo M Morandi

http://www.w3.org/2001/XMLSchema#string

Xifu Shang

http://www.w3.org/2001/XMLSchema#string

Yongjun Wang

http://www.w3.org/2001/XMLSchema#string

Yufeng Dong

http://www.w3.org/2001/XMLSchema#string

Zhengliang Luo

http://www.w3.org/2001/XMLSchema#string

P2860

Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

CDK inhibitors: positive and negative regulators of G1-phase progression

p21 is a universal inhibitor of cyclin kinases

p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest

Cloning of p57KIP2, a cyclin-dependent kinase inhibitor with unique domain structure and tissue distribution

p57KIP2, a structurally distinct member of the p21CIP1 Cdk inhibitor family, is a candidate tumor suppressor gene

BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation

Cyclin-dependent kinases: engines, clocks, and microprocessors

Developmental regulation of the growth plate

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srep25594

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25594

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scholarly article

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10.1038/SREP25594

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English

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http://www.w3.org/2001/XMLSchema#string

P50

Matthew J Hilton

P577

2016-05-05T00:00:00Z

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27146698

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4857138

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Notch signaling indirectly promotes chondrocyte hypertrophy via regulation of BMP signaling and cell cycle arrest.

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P2860

P2860

Notch signaling indirectly promotes chondrocyte hypertrophy via regulation of BMP signaling and cell cycle arrest.

description

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type

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P1433

P1476

P2093

P2860

P2888

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P356

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P577

P698

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P356

P1433

P1476

P2093

P2860

P2888

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P407

P478

P50

P577

P698

P932