Mouse mammary tumor virus proviruses in T-cell lymphomas lack a negative regulatory element in the long terminal repeat
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Endogenous MMTV proviruses induce susceptibility to both viral and bacterial pathogensHomeoproteins CDP and SATB1 interact: potential for tissue-specific regulationAltered mouse mammary tumor virus transcript synthesis in T-cell lymphoma cells.Mutational and functional analysis of the C-terminal region of the C3H mouse mammary tumor virus superantigenMouse mammary tumor virus sequences responsible for activating cellular oncogenes.The c-myc locus is a common integration site in type B retrovirus-induced T-cell lymphomas.CDP is a repressor of mouse mammary tumor virus expression in the mammary gland.C3H mouse mammary tumor virus superantigen function requires a splice donor site in the envelope gene.Type B leukemogenic virus has a T-cell-specific enhancer that binds AML-1.Conversion of mouse mammary tumor virus to a lymphomagenic virus.The homeodomain protein CDP regulates mammary-specific gene transcription and tumorigenesis.CDP binding to multiple sites in the mouse mammary tumor virus long terminal repeat suppresses basal and glucocorticoid-induced transcriptionThe type B leukemogenic virus truncated superantigen is dispensable for T-cell lymphomagenesis.Selection for c-myc integration sites in polyclonal T-cell lymphomas.Functional redundancy of octamer elements in the mouse mammary tumor virus promoter.Factors controlling the expression of mouse mammary tumour virus.The long terminal repeat region of the mouse mammary tumour virus contains multiple regulatory elementsTissue-specific and ubiquitous factors binding next to the glucocorticoid receptor modulate transcription from the mouse mammary tumor virus promoterStimulation of basal transcription from the mouse mammary tumor virus promoter by Oct proteins.A redundant nuclear protein binding site contributes to negative regulation of the mouse mammary tumor virus long terminal repeat.Cooperation between structural elements in hormono-regulated transcription from the mouse mammary tumor virus promoter.Regulated expression of mouse mammary tumor proviral genes in cells of the B lineage.Nucleoprotein structure influences the response of the mouse mammary tumor virus promoter to activation of the cyclic AMP signalling pathway.The matrix attachment region-binding protein SATB1 participates in negative regulation of tissue-specific gene expression.Mouse mammary tumor virus with rearranged long terminal repeats causes murine lymphomas.A mouse mammary tumor virus promoter element near the transcription initiation siteStably integrated mouse mammary tumor virus long terminal repeat DNA requires the octamer motifs for basal promoter activity.A mouse mammary tumor virus mammary gland enhancer confers tissue-specific but not lactation-dependent expression in transgenic mice.Identification of a novel mammary cell line-specific enhancer element in the long terminal repeat of mouse mammary tumor virus, which interacts with its hormone-responsive elementTwo regions of the mouse mammary tumor virus long terminal repeat regulate the activity of its promoter in mammary cell linesExpression pattern of mouse mammary tumor virus in transgenic mice carrying exogenous proviruses of different originsLipopolysaccharide and dexamethasone induce mouse mammary tumor proviral gene expression and differentiation in B lymphocytes through distinct regulatory pathwaysNegative regulation in correct tissue-specific expression of mouse mammary tumor virus in transgenic miceAn activation-dependent, T-lymphocyte-specific transcriptional activator in the mouse mammary tumor virus env gene.Rev and Rex proteins of human complex retroviruses function with the MMTV Rem-responsive element.Lessons Learned from Mouse Mammary Tumor Virus in Animal Models.Ku antigen-DNA conformation determines the activation of DNA-dependent protein kinase and DNA sequence-directed repression of mouse mammary tumor virus transcription.Identification of Notch1 as a frequent target for provirus insertional mutagenesis in T-cell lymphomas induced by leukemogenic mutants of mouse mammary tumor virus.Extra mouse mammary tumor proviruses in DBA/2 mouse lymphomas acquire a selective advantage in lymphocytes by alteration in the U3 region of the long terminal repeat.Promoter complex in the central part of the mouse mammary tumor virus long terminal repeat.
P2860
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P2860
Mouse mammary tumor virus proviruses in T-cell lymphomas lack a negative regulatory element in the long terminal repeat
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Mouse mammary tumor virus prov ...... nt in the long terminal repeat
@en
type
label
Mouse mammary tumor virus prov ...... nt in the long terminal repeat
@en
prefLabel
Mouse mammary tumor virus prov ...... nt in the long terminal repeat
@en
P2093
P2860
P1433
P1476
Mouse mammary tumor virus prov ...... nt in the long terminal repeat
@en
P2093
P2860
P304
P407
P577
1988-12-01T00:00:00Z