Genetic polymorphism at codon 546 of the human RAD17 contributes to the risk for esophageal squamous cell carcinoma.

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Genetic polymorphism at codon 546 of the human RAD17 contributes to the risk for esophageal squamous cell carcinoma.

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http://www.wikidata.org/entity/Q36874959

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2016 nî lūn-bûn

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2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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Genetic polymorphism at codon ...... ageal squamous cell carcinoma.

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Genetic polymorphism at codon ...... ageal squamous cell carcinoma.

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prefLabel

Genetic polymorphism at codon ...... ageal squamous cell carcinoma.

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P1476

Genetic polymorphism at codon ...... ageal squamous cell carcinoma.

@en

P2093

Akiko Sakai

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Hiroshi Katayama

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Kenji Shimizu

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Sachio Ito

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Shunsuke Tanabe

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Takayuki Sonoyama

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Yasuhiro Shirakawa

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Yoshio Naomoto

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Yuichiro Mita

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Yukiko Yasuda

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P2860

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ATR/ATM-mediated phosphorylation of human Rad17 is required for genotoxic stress responses

Genetic susceptibility to esophageal cancer: the role of the nucleotide excision repair pathway.

Effects of acute ethanol treatment on NCCIT cells and NCCIT cell-derived embryoid bodies (EBs).

HRad17, a human homologue of the Schizosaccharomyces pombe checkpoint gene rad17, is overexpressed in colon carcinoma.

Dial 9-1-1 for DNA damage: the Rad9-Hus1-Rad1 (9-1-1) clamp complex.

DNA damage checkpoints in mammals.

Genetic polymorphisms and esophageal cancer risk.

Alcohol drinking and esophageal cancer risk: an evaluation based on a systematic review of epidemiologic evidence among the Japanese population.

Cigarette smoking and esophageal cancer risk: an evaluation based on a systematic review of epidemiologic evidence among the Japanese population.

P304

58-66

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scholarly article

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P577

2016-03-23T00:00:00Z

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27186329

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P932

4858617

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