Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue. - Wikidata - wikimedia - TriplyDB

Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.

Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.

http://www.wikidata.org/entity/Q36876889

about

Pituitary gland development and disease: from stem cell to hormone production DLK1/PREF1 regulates nutrient metabolism and protects from steatosis.Genetic regulation of murine pituitary development.Developmental and Functional Effects of Steroid Hormones on the Neuroendocrine Axis and Spinal Cord Delta-like protein 1 in the pituitary-adipose axis in the adult male mouse.Regulation of pituitary stem cells by epithelial to mesenchymal transition events and signaling pathways.Role of protein delta homolog 1 in the proliferation and differentiation of ameloblasts.Reduced salivary gland size and increased presence of epithelial progenitor cells in DLK1-deficient mice.Paternally Inherited DLK1 Deletion Associated With Familial Central Precocious Puberty.The effect of delta-like 1 homologue on the proliferation and odontoblastic differentiation in human dental pulp stem cells.Temporal effects of Notch signaling and potential cooperation with multiple downstream effectors on adenohypophysis cell specification in zebrafish.

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P2860

Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.

http://www.wikidata.org/entity/Q36876889

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.

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Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.

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Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.

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Journal of Neuroendocrinology

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Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue

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R Lovell-Badge

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P2860

Selective loss of MEG3 expression and intergenic differentially methylated region hypermethylation in the MEG3/DLK1 locus in human clinically nonfunctioning pituitary adenomas

FA1 immunoreactivity in endocrine tumours and during development of the human fetal pancreas; negative correlation with glucagon expression

Cell proliferation and vascularization in mouse models of pituitary hormone deficiency

Sustained Notch signaling in progenitors is required for sequential emergence of distinct cell lineages during organogenesis

SOX2-expressing progenitor cells generate all of the major cell types in the adult mouse pituitary gland.

Premature differentiation and aberrant movement of pituitary cells lacking both Hes1 and Prop1

The canonical Notch signaling pathway: unfolding the activation mechanism.

Placental development: lessons from mouse mutants

FA1 immunoreactivity in endocrine tumours and during development of the human fetal pancreas; negative correlation with glucagon expression

Pituitary growth hormone network responses are sexually dimorphic and regulated by gonadal steroids in adulthood.

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391-401

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10.1111/JNE.12010

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4

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25

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Paul Le Tissier

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