Lung EC-SOD overexpression attenuates hypoxic induction of Egr-1 and chronic hypoxic pulmonary vascular remodeling. - Wikidata - wikimedia - TriplyDB

Lung EC-SOD overexpression attenuates hypoxic induction of Egr-1 and chronic hypoxic pulmonary vascular remodeling.

Lung EC-SOD overexpression attenuates hypoxic induction of Egr-1 and chronic hypoxic pulmonary vascular remodeling.

http://www.wikidata.org/entity/Q36877360

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Aberrant Pulmonary Vascular Growth and Remodeling in Bronchopulmonary Dysplasia Epigenetics and miRNA emerge as key regulators of smooth muscle cell phenotype and function Disease progression mediated by egr-1 associated signaling in response to oxidative stress Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications N-n-butyl haloperidol iodide protects cardiac microvascular endothelial cells from hypoxia/reoxygenation injury by down-regulating Egr-1 expression Enhanced depolarization-induced pulmonary vasoconstriction following chronic hypoxia requires EGFR-dependent activation of NAD(P)H oxidase 2 Sex-dependent genetic effects on immune responses to a parasitic nematode.Peroxisome proliferator-activated receptor-g agonist treatment increases septation and angiogenesis and decreases airway hyperresponsiveness in a model of experimental neonatal chronic lung disease.Egr-1 expression during neointimal development in flow-associated pulmonary hypertension.Exposure of mice to chronic hypoxia attenuates pulmonary arterial contractile responses to acute hypoxia by increases in extracellular hydrogen peroxide Chronic hypoxia limits H2O2-induced inhibition of ASIC1-dependent store-operated calcium entry in pulmonary arterial smooth muscle.Superoxide dismutases: role in redox signaling, vascular function, and diseases.The role of redox changes in oxygen sensing.Selective depletion of vascular EC-SOD augments chronic hypoxic pulmonary hypertension A common polymorphism in extracellular superoxide dismutase affects cardiopulmonary disease risk by altering protein distribution.SOD mRNA and MDA expression in rectus femoris muscle of rats with different eccentric exercise programs and time points Extracellular superoxide dismutase regulates the expression of small gtpase regulatory proteins GEFs, GAPs, and GDI.Chronic hypoxia promotes pulmonary artery endothelial cell proliferation through H2O2-induced 5-lipoxygenase.Altered pulmonary artery endothelial-smooth muscle cell interactions in experimental congenital diaphragmatic hernia.Exacerbated pulmonary arterial hypertension and right ventricular hypertrophy in animals with loss of function of extracellular superoxide dismutase.Intermittent hypoxia augments pulmonary vascular smooth muscle reactivity to NO: regulation by reactive oxygen species Xanthine oxidase-derived ROS upregulate Egr-1 via ERK1/2 in PA smooth muscle cells; model to test impact of extracellular ROS in chronic hypoxia.Histone acetylation regulates the cell-specific and interferon-γ-inducible expression of extracellular superoxide dismutase in human pulmonary arteries.Effect of N-n-butyl haloperidol iodide on ROS/JNK/Egr-1 signaling in H9c2 cells after hypoxia/reoxygenation.NADPH oxidase-derived ROS and the regulation of pulmonary vessel tone Bone morphogenetic protein receptor type II deficiency and increased inflammatory cytokine production. A gateway to pulmonary arterial hypertension PGC-1α induction in pulmonary arterial hypertension Lack of EC-SOD worsens alveolar and vascular development in a neonatal mouse model of bleomycin-induced bronchopulmonary dysplasia and pulmonary hypertension.Superoxide dismutase mimetic, MnTE-2-PyP, attenuates chronic hypoxia-induced pulmonary hypertension, pulmonary vascular remodeling, and activation of the NALP3 inflammasome.Activation of Calpain-2 by Mediators in Pulmonary Vascular Remodeling of Pulmonary Arterial Hypertension.NFAT is required for spontaneous pulmonary hypertension in superoxide dismutase 1 knockout mice.Pulmonary hypertension secondary to left-heart failure involves peroxynitrite-induced downregulation of PTEN in the lung.Histone deacetylation contributes to low extracellular superoxide dismutase expression in human idiopathic pulmonary arterial hypertension.Current concepts on the role of inflammation in COPD and lung cancer.Reactive oxygen and nitrogen species in pulmonary hypertension.Pulmonary artery endothelial cell dysfunction and decreased populations of highly proliferative endothelial cells in experimental congenital diaphragmatic hernia.N-n-butyl Haloperidol Iodide Protects against Hypoxia/Reoxygenation Injury in Cardiac Microvascular Endothelial Cells by Regulating the ROS/MAPK/Egr-1 Pathway.Current concepts on oxidative/carbonyl stress, inflammation and epigenetics in pathogenesis of chronic obstructive pulmonary disease.Lung oxidative damage by hypoxia Reactive oxygen species as therapeutic targets in pulmonary hypertension.

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P2860

Lung EC-SOD overexpression attenuates hypoxic induction of Egr-1 and chronic hypoxic pulmonary vascular remodeling.

http://www.wikidata.org/entity/Q36877360

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Lung EC-SOD overexpression att ...... pulmonary vascular remodeling.

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type

label

Lung EC-SOD overexpression att ...... pulmonary vascular remodeling.

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prefLabel

Lung EC-SOD overexpression att ...... pulmonary vascular remodeling.

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AJPLUNG.90293.2008

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Lung EC-SOD overexpression att ...... pulmonary vascular remodeling.

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Eva Nozik-Grayck

http://www.w3.org/2001/XMLSchema#string

Hagir B Suliman

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Joseph Albietz

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Kevin Roush

http://www.w3.org/2001/XMLSchema#string

Kurt R Stenmark

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Susan Majka

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Zachary Van Rheen

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P2860

NAB2, a corepressor of NGFI-A (Egr-1) and Krox20, is induced by proliferative and differentiative stimuli

Overexpression of extracellular superoxide dismutase reduces acute radiation induced lung toxicity

Superoxide dismutase-3 promotes full expression of the EPO response to hypoxia

Hypoxia/Hypoxemia-Induced activation of the procoagulant pathways and the pathogenesis of ischemia-associated thrombosis.

Extracellular superoxide dismutase in the airways of transgenic mice reduces inflammation and attenuates lung toxicity following hyperoxia

Extracellular superoxide dismutase.

Egr-1 induces the expression of its corepressor nab2 by activation of the nab2 promoter thereby establishing a negative feedback loop.

Early growth response-1 in cardiovascular pathobiology.

Hypoxia-induced pulmonary vascular remodeling: cellular and molecular mechanisms.

Apical membrane targeting and trafficking of the human proton-coupled transporter in polarized epithelia.

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