Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells - Wikidata - wikimedia - TriplyDB

Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

http://www.wikidata.org/entity/Q36879043

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Reserve stem cells: Differentiated cells reprogram to fuel repair, metaplasia, and neoplasia in the adult gastrointestinal tract Silencing Mist1 Gene Expression Is Essential for Recovery from Acute Pancreatitis Epigenetic reprogramming in Mist1(-/-) mice predicts the molecular response to cerulein-induced pancreatitis Interleukin-6 is required for pancreatic cancer progression by promoting MAPK signaling activation and oxidative stress resistance RAB26 coordinates lysosome traffic and mitochondrial localization.Identification of key transcription factors in caerulein-induced pancreatitis through expression profiling data.MAPK signaling is required for dedifferentiation of acinar cells and development of pancreatic intraepithelial neoplasia in mice.The basic helix-loop-helix transcription factor E47 reprograms human pancreatic cancer cells to a quiescent acinar state with reduced tumorigenic potential.The ubiquitin ligase Mindbomb 1 coordinates gastrointestinal secretory cell maturation MIST1 Links Secretion and Stress as Both Target and Regulator of the UPR.MIST1 and PTF1 Collaborate in Feed-forward Regulatory Loops that Maintain the Pancreatic Acinar Phenotype in Adult Mice.A single transcription factor is sufficient to induce and maintain secretory cell architecture Pancreatic adaptive responses in alcohol abuse: Role of the unfolded protein response.Neat1 is a p53-inducible lincRNA essential for transformation suppression.Acinar cell plasticity and development of pancreatic ductal adenocarcinoma.The big and intricate dreams of little organelles: Embracing complexity in the study of membrane traffic.Plasticity and dedifferentiation within the pancreas: development, homeostasis, and disease.The basic helix-loop-helix transcription factor, Mist1, induces maturation of mouse fetal hepatoblasts.Transcriptional Maintenance of Pancreatic Acinar Identity, Differentiation, and Homeostasis by PTF1A.Change isn't always better.Cellular and molecular mechanisms coordinating pancreas development.Regenerative proliferation of differentiated cells by mTORC1-dependent paligenosis.Encapsulation of primary salivary gland cells in enzymatically degradable poly(ethylene glycol) hydrogels promotes acinar cell characteristics.Off and back-on again: a tumor suppressor's tale.Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment.Stabilization of HIF-2α impacts pancreas growth

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P2860

Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

http://www.wikidata.org/entity/Q36879043

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

@yue

2012年論文

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2012年論文

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2012年論文

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2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

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type

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Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

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Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

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P356

J.GASTRO.2012.04.011

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Gastroenterology

P1476

Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells

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P2093

Barbara Damsz

http://www.w3.org/2001/XMLSchema#string

Brett Marshall

http://www.w3.org/2001/XMLSchema#string

Chirayu Goswami

http://www.w3.org/2001/XMLSchema#string

Daniel Direnzo

http://www.w3.org/2001/XMLSchema#string

David A Hess

http://www.w3.org/2001/XMLSchema#string

Judy E Hallett

http://www.w3.org/2001/XMLSchema#string

Raymond J Macdonald

http://www.w3.org/2001/XMLSchema#string

Stephen F Konieczny

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Yunlong Liu

http://www.w3.org/2001/XMLSchema#string

P2860

RAB26 and RAB3D are direct transcriptional targets of MIST1 that regulate exocrine granule maturation

The basic helix-loop-helix transcription factor Mist1 functions as a transcriptional repressor of myoD

Mature chief cells are cryptic progenitors for metaplasia in the stomach

The bHLH transcription factor Mist1 is required to maintain exocrine pancreas cell organization and acinar cell identity

The gastric epithelial progenitor cell niche and differentiation of the zymogenic (chief) cell lineage.

The maturation of mucus-secreting gastric epithelial progenitors into digestive-enzyme secreting zymogenic cells requires Mist1

Ubiquitin fold modifier 1 (UFM1) and its target UFBP1 protect pancreatic beta cells from ER stress-induced apoptosis

Aberrant localization of intracellular organelles, Ca2+ signaling, and exocytosis in Mist1 null mice

Exocrine specific expression of Connexin32 is dependent on the basic helix-loop-helix transcription factor Mist1

Transcriptional orchestration of the regulated secretory pathway in neurons by the bHLH protein DIMM

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469-480

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10.1053/J.GASTRO.2012.04.011

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