Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells
about
Reserve stem cells: Differentiated cells reprogram to fuel repair, metaplasia, and neoplasia in the adult gastrointestinal tractSilencing Mist1 Gene Expression Is Essential for Recovery from Acute PancreatitisEpigenetic reprogramming in Mist1(-/-) mice predicts the molecular response to cerulein-induced pancreatitisInterleukin-6 is required for pancreatic cancer progression by promoting MAPK signaling activation and oxidative stress resistanceRAB26 coordinates lysosome traffic and mitochondrial localization.Identification of key transcription factors in caerulein-induced pancreatitis through expression profiling data.MAPK signaling is required for dedifferentiation of acinar cells and development of pancreatic intraepithelial neoplasia in mice.The basic helix-loop-helix transcription factor E47 reprograms human pancreatic cancer cells to a quiescent acinar state with reduced tumorigenic potential.The ubiquitin ligase Mindbomb 1 coordinates gastrointestinal secretory cell maturationMIST1 Links Secretion and Stress as Both Target and Regulator of the UPR.MIST1 and PTF1 Collaborate in Feed-forward Regulatory Loops that Maintain the Pancreatic Acinar Phenotype in Adult Mice.A single transcription factor is sufficient to induce and maintain secretory cell architecturePancreatic adaptive responses in alcohol abuse: Role of the unfolded protein response.Neat1 is a p53-inducible lincRNA essential for transformation suppression.Acinar cell plasticity and development of pancreatic ductal adenocarcinoma.The big and intricate dreams of little organelles: Embracing complexity in the study of membrane traffic.Plasticity and dedifferentiation within the pancreas: development, homeostasis, and disease.The basic helix-loop-helix transcription factor, Mist1, induces maturation of mouse fetal hepatoblasts.Transcriptional Maintenance of Pancreatic Acinar Identity, Differentiation, and Homeostasis by PTF1A.Change isn't always better.Cellular and molecular mechanisms coordinating pancreas development.Regenerative proliferation of differentiated cells by mTORC1-dependent paligenosis.Encapsulation of primary salivary gland cells in enzymatically degradable poly(ethylene glycol) hydrogels promotes acinar cell characteristics.Off and back-on again: a tumor suppressor's tale.Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment.Stabilization of HIF-2α impacts pancreas growth
P2860
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P2860
Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells
@en
type
label
Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells
@en
prefLabel
Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells
@en
P2093
P2860
P1433
P1476
Induced Mist1 expression promotes remodeling of mouse pancreatic acinar cells
@en
P2093
Barbara Damsz
Brett Marshall
Chirayu Goswami
Daniel Direnzo
David A Hess
Judy E Hallett
Raymond J Macdonald
Stephen F Konieczny
Yunlong Liu
P2860
P304
P356
10.1053/J.GASTRO.2012.04.011
P407
P50
P577
2012-04-14T00:00:00Z