CD8(+) granzyme B(+)-mediated tissue injury vs. CD4(+)IFNγ(+)-mediated parasite killing in human cutaneous leishmaniasis - Wikidata - wikimedia - TriplyDB

CD8(+) granzyme B(+)-mediated tissue injury vs. CD4(+)IFNγ(+)-mediated parasite killing in human cutaneous leishmaniasis

CD8(+) granzyme B(+)-mediated tissue injury vs. CD4(+)IFNγ(+)-mediated parasite killing in human cutaneous leishmaniasis

http://www.wikidata.org/entity/Q36886086

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Post-Genomics and Vaccine Improvement for Leishmania Kinetoplastid Membrane Protein-11 as a Vaccine Candidate and a Virulence Factor in Leishmania Natural Products: Insights into Leishmaniasis Inflammatory Response Immunoregulation in human American leishmaniasis: balancing pathology and protection Regulation of immunity during visceral Leishmania infection Asymptomatic infection with American cutaneous leishmaniasis: epidemiological and immunological studies In vitro evaluation of a soluble Leishmania promastigote surface antigen as a potential vaccine candidate against human leishmaniasis CD3+CD4negCD8neg (double negative) T lymphocytes and NKT cells as the main cytotoxic-related-CD107a+ cells in lesions of cutaneous leishmaniasis caused by Leishmania (Viannia) braziliensis.A secondary wave of neutrophil infiltration causes necrosis and ulceration in lesions of experimental American cutaneous leishmaniasis CD8+ T cells in situ in different clinical forms of human cutaneous leishmaniasis.Comparative evaluation of lesion development, tissue damage, and cytokine expression in golden hamsters (Mesocricetus auratus) infected by inocula with different Leishmania (Viannia) braziliensis concentrations.Genomic profiling of human Leishmania braziliensis lesions identifies transcriptional modules associated with cutaneous immunopathology The immunological, environmental, and phylogenetic perpetrators of metastatic leishmaniasis.Granzyme-mediated regulation of host defense in the liver in experimental Leishmania donovani infection.Protective and pathological functions of CD8+ T cells in Leishmania braziliensis infection.Intermediate monocytes contribute to pathologic immune response in Leishmania braziliensis infections.Engagement of NKG2D on bystander memory CD8 T cells promotes increased immunopathology following Leishmania major infection Apoptosis and frequency of total and effector CD8+ T lymphocytes from cutaneous leishmaniasis patients during antimonial therapy.Prediction of CD8+ Epitopes in Leishmania braziliensis Proteins Using EPIBOT: In Silico Search and In Vivo Validation.Immune response to Leishmania antigens in an AIDS patient with mucocutaneous leishmaniasis as a manifestation of immune reconstitution inflammatory syndrome (IRIS): a case report The Immunology of a Healing Response in Cutaneous Leishmaniasis Treated with Localized Heat or Systemic Antimonial Therapy.T-cell receptor Vβ repertoire of CD8+ T-lymphocyte subpopulations in cutaneous leishmaniasis patients from the state of Rio de Janeiro, Brazil.Lymphocytic Choriomeningitis Virus Expands a Population of NKG2D+CD8+ T Cells That Exacerbates Disease in Mice Coinfected with Leishmania major.Meta-transcriptome Profiling of the Human-Leishmania braziliensis Cutaneous Lesion.CD8+ T cell cytotoxicity mediates pathology in the skin by inflammasome activation and IL-1β production.Cutaneous Infection with Leishmania major Mediates Heterologous Protection against Visceral Infection with Leishmania infantum A Potential Role for Mononuclear Phagocytes in Cutaneous Ulcer Development in Human Immunodeficiency Virus-Leishmania braziliensis Coinfection.CD8 T cell exhaustion in human visceral leishmaniasis.Characterization of the Histopathologic Features in Patients in the Early and Late Phases of Cutaneous Leishmaniasis.The Role of CD4 and CD8 T Cells in Human Cutaneous Leishmaniasis.CD8+ T cells in cutaneous leishmaniasis: the good, the bad, and the ugly.Memory T cells in cutaneous leishmaniasis.Immunopathological characterization of human cutaneous leishmaniasis lesions caused by Leishmania (Viannia) spp. in Amazonian Brazil.American cutaneous leishmaniasis: In situ immune response of patients with recent and late lesions.The Cytotoxic T Lymphocyte Antigen-4 +49A/G Single Nucleotide Polymorphism Association With Visceral Leishmaniasis.Cutaneous Leishmaniasis Induces a Transmissible Dysbiotic Skin Microbiota that Promotes Skin Inflammation.Leishmania major large RAB GTPase is highly immunogenic in individuals immune to cutaneous and visceral leishmaniasis.Immunologic Markers of Protection in Leishmania (Viannia) braziliensis Infection: A 5-Year Cohort Study Effector memory CD4(+) T cells differentially express activation associated molecules depending on the duration of American cutaneous leishmaniasis lesions.Evaluation of cellular immunological responses in mono- and polymorphic clinical forms of post-kala-azar dermal leishmaniasis in India.

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CD8(+) granzyme B(+)-mediated tissue injury vs. CD4(+)IFNγ(+)-mediated parasite killing in human cutaneous leishmaniasis

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name

CD8(+) granzyme B(+)-mediated ...... human cutaneous leishmaniasis

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CD8(+) granzyme B(+)-mediated ...... human cutaneous leishmaniasis

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CD8(+) granzyme B(+)-mediated ...... human cutaneous leishmaniasis

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Journal of Investigative Dermatology

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CD8(+) granzyme B(+)-mediated ...... human cutaneous leishmaniasis

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Aldina Barral

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Almério Noronha

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Claire da Silva Santos

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Cláudia Ida Brodskyn

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Cristina Ribeiro Cardoso

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Jackson Costa

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Morgana J Lordelo

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Viviane Boaventura

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P2860

The influence of early neutrophil-Leishmania interactions on the host immune response to infection

Platelets induce apoptosis during sepsis in a contact-dependent manner that is inhibited by GPIIb/IIIa blockade

CD8+ T cells as a source of IFN-γ production in human cutaneous leishmaniasis.

CD8 cells of patients with diffuse cutaneous leishmaniasis display functional exhaustion: the latter is reversed, in vitro, by TLR2 agonists.

TLR1/2 activation during heterologous prime-boost vaccination (DNA-MVA) enhances CD8+ T Cell responses providing protection against Leishmania (Viannia)

Differences in gamma interferon production in vitro predict the pace of the in vivo response to Leishmania amazonensis in healthy volunteers.

Regulation of CD8+ T cell responses to infection with parasitic protozoa.

Up-regulation of Th1-type responses in mucosal leishmaniasis patients

Granzyme B: a natural born killer.

Chemoattractants and their receptors in homeostasis and inflammation.

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1533-1540

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10.1038/JID.2013.4

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6

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133

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Manoel Barral-Netto

Johan Van Weyenbergh

Valeria M Borges

Camila Indiani de Oliveira

Natalia Machado Tavares

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2013-01-15T00:00:00Z

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234142116

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23321919

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