Expression of herpes simplex virus type 1 major DNA-binding protein, ICP8, in transformed cell lines: complementation of deletion mutants and inhibition of wild-type virus.
about
Roscovitine inhibits activation of promoters in herpes simplex virus type 1 genomes independently of promoter-specific factorsRegulated expression of a Sindbis virus replicon by herpesvirus promoters.A tumor host range selection procedure identifies p150(sal2) as a target of polyoma virus large T antigen.Aphidicolin resistance in herpes simplex virus type I reveals features of the DNA polymerase dNTP binding site.A cytomegalovirus protein with properties of herpes simplex virus ICP8: partial purification of the polypeptide and map position of the geneRetention of the herpes simplex virus type 1 (HSV-1) UL37 protein on single-stranded DNA columns requires the HSV-1 ICP8 protein.Intragenic complementation of herpes simplex virus ICP8 DNA-binding protein mutants.Isolation of a herpes simplex virus type 1 mutant deleted for the essential UL42 gene and characterization of its null phenotype.Surface lysine and tyrosine residues are required for interaction of the major herpes simplex virus type 1 DNA-binding protein with single-stranded DNA.Potential role for herpes simplex virus ICP8 DNA replication protein in stimulation of late gene expressionHerpes simplex virus type 1 ICP27 deletion mutants exhibit altered patterns of transcription and are DNA deficientIntragenic complementation among partial peptides of herpes simplex virus regulatory protein ICP4.The herpes simplex virus type 1 UL42 gene product: a subunit of DNA polymerase that functions to increase processivity.Characterization of a major DNA-binding domain in the herpes simplex virus type 1 DNA-binding protein (ICP8).Identification of a site on herpes simplex virus type 1 glycoprotein D that is essential for infectivityGenetic evidence for multiple nuclear functions of the herpes simplex virus ICP8 DNA-binding proteinPhysical and functional domains of the herpes simplex virus transcriptional regulatory protein ICP4.The herpes simplex virus origins of DNA synthesis in the S component are each contained in a transcribed open reading frame.Herpes simplex virus type 1 oriL is not required for virus replication or for the establishment and reactivation of latent infection in mice.Distal protein sequences can affect the function of a nuclear localization signalPseudorabies virus protein homologous to herpes simplex virus type 1 ICP18.5 is necessary for capsid maturation.Deletions of the carboxy terminus of herpes simplex virus type 1 UL42 define a conserved amino-terminal functional domain.A subset of herpes simplex virus replication genes provides helper functions for productive adeno-associated virus replication.A subset of herpes simplex virus replication genes induces DNA amplification within the host cell genome.
P2860
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P2860
Expression of herpes simplex virus type 1 major DNA-binding protein, ICP8, in transformed cell lines: complementation of deletion mutants and inhibition of wild-type virus.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Expression of herpes simplex v ...... inhibition of wild-type virus.
@en
type
label
Expression of herpes simplex v ...... inhibition of wild-type virus.
@en
prefLabel
Expression of herpes simplex v ...... inhibition of wild-type virus.
@en
P2860
P1433
P1476
Expression of herpes simplex v ...... inhibition of wild-type virus
@en
P2093
P A Schaffer
P K Orberg
P2860
P304
P407
P577
1987-04-01T00:00:00Z