Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C
about
Targeting of adenosine receptors in ischemia-reperfusion injury.AMP579 is revealed to be a potent A2b-adenosine receptor agonist in human 293 cells and rabbit hearts.The critical role of intracellular zinc in adenosine A(2) receptor activation induced cardioprotection against reperfusion injuryAdenosine receptor-mediated cardioprotection: are all 4 subtypes required or redundant?Cardioprotective PKG-independent NO signaling at reperfusionMitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis.Adenosine A₂A and A₂B receptors are both required for adenosine A₁ receptor-mediated cardioprotection.Transgenic over expression of ectonucleotide triphosphate diphosphohydrolase-1 protects against murine myocardial ischemic injury.Adora2b signaling on bone marrow derived cells dampens myocardial ischemia-reperfusion injury.Adenosine augments IL-10 production by microglial cells through an A2B adenosine receptor-mediated processDifferential Tissue-Specific Function of Adora2b in Cardioprotection.Influenza A Virus-Induced Expression of a GalNAc Transferase, GALNT3, via MicroRNAs Is Required for Enhanced Viral Replication.Adenosine A2A and A2B receptors work in concert to induce a strong protection against reperfusion injury in rat hearts.Mechanism of cardioprotection by early ischemic preconditioningAdenosine receptor targeting in health and disease.A2 adenosine receptors and vascular pathologies.Targeting reperfusion injury in acute myocardial infarction: a review of reperfusion injury pharmacotherapy.Signalling pathways and mechanisms of protection in pre- and postconditioning: historical perspective and lessons for the future.The "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle.ATP-induced cardioprotection against myocardial ischemia/reperfusion injury is mediated through the RISK pathway.BAY 58-2667, a nitric oxide-independent guanylyl cyclase activator, pharmacologically post-conditions rabbit and rat hearts.Insulin and GSK3β-inhibition abrogates the infarct sparing-effect of ischemic postconditioning in ex vivo rat hearts.The role of cGMP and PKG in cardioprotection.
P2860
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P2860
Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Infarct limitation by a protei ...... b agonists by protein kinase C
@en
type
label
Infarct limitation by a protei ...... b agonists by protein kinase C
@en
prefLabel
Infarct limitation by a protei ...... b agonists by protein kinase C
@en
P2093
P2860
P1476
Infarct limitation by a protei ...... b agonists by protein kinase C
@en
P2093
James M Downey
Michael V Cohen
Nataliya V Solenkova
Turhan Dost
Victoriya Solodushko
Xi-Ming Yang
Yanping Liu
P2860
P304
H1288-H1295
P356
10.1152/AJPHEART.00209.2008
P50
P577
2008-07-25T00:00:00Z