Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C - Wikidata - wikimedia - TriplyDB

Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

http://www.wikidata.org/entity/Q36893258

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Targeting of adenosine receptors in ischemia-reperfusion injury.AMP579 is revealed to be a potent A2b-adenosine receptor agonist in human 293 cells and rabbit hearts.The critical role of intracellular zinc in adenosine A(2) receptor activation induced cardioprotection against reperfusion injury Adenosine receptor-mediated cardioprotection: are all 4 subtypes required or redundant?Cardioprotective PKG-independent NO signaling at reperfusion Mitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis.Adenosine A₂A and A₂B receptors are both required for adenosine A₁ receptor-mediated cardioprotection.Transgenic over expression of ectonucleotide triphosphate diphosphohydrolase-1 protects against murine myocardial ischemic injury.Adora2b signaling on bone marrow derived cells dampens myocardial ischemia-reperfusion injury.Adenosine augments IL-10 production by microglial cells through an A2B adenosine receptor-mediated process Differential Tissue-Specific Function of Adora2b in Cardioprotection.Influenza A Virus-Induced Expression of a GalNAc Transferase, GALNT3, via MicroRNAs Is Required for Enhanced Viral Replication.Adenosine A2A and A2B receptors work in concert to induce a strong protection against reperfusion injury in rat hearts.Mechanism of cardioprotection by early ischemic preconditioning Adenosine receptor targeting in health and disease.A2 adenosine receptors and vascular pathologies.Targeting reperfusion injury in acute myocardial infarction: a review of reperfusion injury pharmacotherapy.Signalling pathways and mechanisms of protection in pre- and postconditioning: historical perspective and lessons for the future.The "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle.ATP-induced cardioprotection against myocardial ischemia/reperfusion injury is mediated through the RISK pathway.BAY 58-2667, a nitric oxide-independent guanylyl cyclase activator, pharmacologically post-conditions rabbit and rat hearts.Insulin and GSK3β-inhibition abrogates the infarct sparing-effect of ischemic postconditioning in ex vivo rat hearts.The role of cGMP and PKG in cardioprotection.

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P2860

Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

http://www.wikidata.org/entity/Q36893258

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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Infarct limitation by a protei ...... b agonists by protein kinase C

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AJPHEART.00209.2008

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American Journal of Physiology - Heart and Circulatory Physiology

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Infarct limitation by a protei ...... b agonists by protein kinase C

@en

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James M Downey

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Michael V Cohen

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Nataliya V Solenkova

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Turhan Dost

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Victoriya Solodushko

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Xi-Ming Yang

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Yanping Liu

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P2860

Characterization of human A(2B) adenosine receptors: radioligand binding, western blotting, and coupling to G(q) in human embryonic kidney 293 cells and HMC-1 mast cells

Signalling via the reperfusion injury signalling kinase (RISK) pathway links closure of the mitochondrial permeability transition pore to cardioprotection.

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

Infarct-sparing effect of myocardial postconditioning is dependent on protein kinase C signalling.

Postconditioning protects rabbit hearts through a protein kinase C-adenosine A2b receptor cascade.

Sildenafil and vardenafil but not nitroglycerin limit myocardial infarction through opening of mitochondrial K(ATP) channels when administered at reperfusion following ischemia in rabbits

Cyclic GMP and protein kinase-G in myocardial ischaemia-reperfusion: opportunities and obstacles for survival signaling.

Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injury

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

Redox signaling at reperfusion is required for protection from ischemic preconditioning but not from a direct PKC activator.

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10.1152/AJPHEART.00209.2008

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3

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295

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51431396

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18660452

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